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Hepatitis C virus immune escape via exploitation of a hole in the T cell repertoire.
Wölfl M, Rutebemberwa A, Mosbruger T, Mao Q, Li HM, Netski D, Ray SC, Pardoll D, Sidney J, Sette A, Allen T, Kuntzen T, Kavanagh DG, Kuball J, Greenberg PD, Cox AL. Wölfl M, et al. J Immunol. 2008 Nov 1;181(9):6435-46. doi: 10.4049/jimmunol.181.9.6435. J Immunol. 2008. PMID: 18941234 Free PMC article.
High-programmed death-1 levels on hepatitis C virus-specific T cells during acute infection are associated with viral persistence and require preservation of cognate antigen during chronic infection.
Rutebemberwa A, Ray SC, Astemborski J, Levine J, Liu L, Dowd KA, Clute S, Wang C, Korman A, Sette A, Sidney J, Pardoll DM, Cox AL. Rutebemberwa A, et al. J Immunol. 2008 Dec 15;181(12):8215-25. doi: 10.4049/jimmunol.181.12.8215. J Immunol. 2008. PMID: 19050238 Free PMC article.