R01 CA095277-06A1/CA/NCI NIH HHS/United States[Grants and Funding] - Search Results

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2025	0

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Eya2 is required to mediate the pro-metastatic functions of Six1 via the induction of TGF-β signaling, epithelial-mesenchymal transition, and cancer stem cell properties.

Farabaugh SM, Micalizzi DS, Jedlicka P, Zhao R, Ford HL. Farabaugh SM, et al. Oncogene. 2012 Feb 2;31(5):552-62. doi: 10.1038/onc.2011.259. Epub 2011 Jun 27. Oncogene. 2012. PMID: 21706047 Free PMC article.

Homeoprotein Six1 increases TGF-beta type I receptor and converts TGF-beta signaling from suppressive to supportive for tumor growth.

Micalizzi DS, Wang CA, Farabaugh SM, Schiemann WP, Ford HL. Micalizzi DS, et al. Cancer Res. 2010 Dec 15;70(24):10371-80. doi: 10.1158/0008-5472.CAN-10-1354. Epub 2010 Nov 5. Cancer Res. 2010. PMID: 21056993 Free PMC article.

The miR-106b-25 cluster targets Smad7, activates TGF-β signaling, and induces EMT and tumor initiating cell characteristics downstream of Six1 in human breast cancer.

Smith AL, Iwanaga R, Drasin DJ, Micalizzi DS, Vartuli RL, Tan AC, Ford HL. Smith AL, et al. Oncogene. 2012 Dec 13;31(50):5162-71. doi: 10.1038/onc.2012.11. Epub 2012 Jan 30. Oncogene. 2012. PMID: 22286770 Free PMC article.

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