K01AR048594/AR/NIAMS NIH HHS/United States[Grants and Funding] - Search Results

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2006	1
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2025	0

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Defective lamin A-Rb signaling in Hutchinson-Gilford Progeria Syndrome and reversal by farnesyltransferase inhibition.

Marji J, O'Donoghue SI, McClintock D, Satagopam VP, Schneider R, Ratner D, Worman HJ, Gordon LB, Djabali K. Marji J, et al. PLoS One. 2010 Jun 15;5(6):e11132. doi: 10.1371/journal.pone.0011132. PLoS One. 2010. PMID: 20559568 Free PMC article.

The mutant form of lamin A that causes Hutchinson-Gilford progeria is a biomarker of cellular aging in human skin.

McClintock D, Ratner D, Lokuge M, Owens DM, Gordon LB, Collins FS, Djabali K. McClintock D, et al. PLoS One. 2007 Dec 5;2(12):e1269. doi: 10.1371/journal.pone.0001269. PLoS One. 2007. PMID: 18060063 Free PMC article.

Hutchinson-Gilford progeria mutant lamin A primarily targets human vascular cells as detected by an anti-Lamin A G608G antibody.

McClintock D, Gordon LB, Djabali K. McClintock D, et al. Proc Natl Acad Sci U S A. 2006 Feb 14;103(7):2154-9. doi: 10.1073/pnas.0511133103. Epub 2006 Feb 6. Proc Natl Acad Sci U S A. 2006. PMID: 16461887 Free PMC article.

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