Toda M - Search Results

1

Immunologic self-tolerance maintained by CD25+CD4+ naturally anergic and suppressive T cells: induction of autoimmune disease by breaking their anergic/suppressive state.

Takahashi T, Kuniyasu Y, Toda M, Sakaguchi N, Itoh M, Iwata M, Shimizu J, Sakaguchi S. Takahashi T, et al. Among authors: toda m. Int Immunol. 1998 Dec;10(12):1969-80. doi: 10.1093/intimm/10.12.1969. Int Immunol. 1998. PMID: 9885918

2

Immunologic tolerance maintained by CD25+ CD4+ regulatory T cells: their common role in controlling autoimmunity, tumor immunity, and transplantation tolerance.

Sakaguchi S, Sakaguchi N, Shimizu J, Yamazaki S, Sakihama T, Itoh M, Kuniyasu Y, Nomura T, Toda M, Takahashi T. Sakaguchi S, et al. Among authors: toda m. Immunol Rev. 2001 Aug;182:18-32. doi: 10.1034/j.1600-065x.2001.1820102.x. Immunol Rev. 2001. PMID: 11722621 Review.

3

Immunologic self-tolerance maintained by activated T cells expressing IL-2 receptor alpha-chains (CD25). Breakdown of a single mechanism of self-tolerance causes various autoimmune diseases.

Sakaguchi S, Sakaguchi N, Asano M, Itoh M, Toda M. Sakaguchi S, et al. Among authors: toda m. J Immunol. 1995 Aug 1;155(3):1151-64. J Immunol. 1995. PMID: 7636184

4

T cell-mediated maintenance of natural self-tolerance: its breakdown as a possible cause of various autoimmune diseases.

Sakaguchi S, Toda M, Asano M, Itoh M, Morse SS, Sakaguchi N. Sakaguchi S, et al. Among authors: toda m. J Autoimmun. 1996 Apr;9(2):211-20. doi: 10.1006/jaut.1996.0026. J Autoimmun. 1996. PMID: 8738965 Review.

5

Autoimmune disease as a consequence of developmental abnormality of a T cell subpopulation.

Asano M, Toda M, Sakaguchi N, Sakaguchi S. Asano M, et al. Among authors: toda m. J Exp Med. 1996 Aug 1;184(2):387-96. doi: 10.1084/jem.184.2.387. J Exp Med. 1996. PMID: 8760792 Free PMC article.

6

Pillars article: immunologic self-tolerance maintained by activated T cells expressing IL-2 receptor α-chains (CD25). Breakdown of a single mechanism of self-tolerance causes various autoimmune diseases. J. Immunol. 1995.

Sakaguchi S, Sakaguchi N, Asano M, Itoh M, Toda M. Sakaguchi S, et al. Among authors: toda m. J Immunol. 2011 Apr 1;186(7):3808-21. J Immunol. 2011. PMID: 21422251 No abstract available.

7

Induction of autoimmune disease in mice by germline alteration of the T cell receptor gene expression.

Sakaguchi S, Ermak TH, Toda M, Berg LJ, Ho W, Fazekas de St Groth B, Peterson PA, Sakaguchi N, Davis MM. Sakaguchi S, et al. Among authors: toda m. J Immunol. 1994 Feb 1;152(3):1471-84. J Immunol. 1994. PMID: 7905503

8

Induction of bronchial hyperresponsiveness by Bordetella bronchiseptica infection in dogs.

Nishikata H, Kobayashi H, Sato H, Okada Y, Adachi M, Takahashi T, Soejima K, Hosono K, Toda M. Nishikata H, et al. Among authors: toda m. Ann Allergy. 1989 Jan;62(1):51-8. Ann Allergy. 1989. PMID: 2643371

9

Down-regulation of antigen-specific antibody production by TCR antagonist peptides in vivo.

Toda M, Totsuka M, Furukawa S, Yokota K, Yoshioka T, Ametani A, Kaminogawa S. Toda M, et al. Eur J Immunol. 2000 Feb;30(2):403-14. doi: 10.1002/1521-4141(200002)30:2<403::AID-IMMU403>3.0.CO;2-8. Eur J Immunol. 2000. PMID: 10671195 Free article.

10

Dietary fiber pectin alters the gut microbiota and diminishes the inflammatory immune responses in an experimental peach allergy mouse model.

Steigerwald H, Albrecht M, Blissenbach B, Krause M, Wangorsch A, Schott M, Gonzalez-Menendez I, Quintanilla-Martinez L, Toda M, Vieths S, Krut O, Scheurer S, Blanco-Pérez F. Steigerwald H, et al. Among authors: toda m. Sci Rep. 2024 Dec 16;14(1):30503. doi: 10.1038/s41598-024-82210-3. Sci Rep. 2024. PMID: 39681664 Free PMC article.

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