Martin GM - Search Results

1

cDNA cloning and chromosome mapping of the human Fe65 gene: interaction of the conserved cytoplasmic domains of the human beta-amyloid precursor protein and its homologues with the mouse Fe65 protein.

Bressler SL, Gray MD, Sopher BL, Hu Q, Hearn MG, Pham DG, Dinulos MB, Fukuchi K, Sisodia SS, Miller MA, Disteche CM, Martin GM. Bressler SL, et al. Among authors: martin gm. Hum Mol Genet. 1996 Oct;5(10):1589-98. doi: 10.1093/hmg/5.10.1589. Hum Mol Genet. 1996. PMID: 8894693

2

A candidate molecular mechanism for the association of an intronic polymorphism of FE65 with resistance to very late onset dementia of the Alzheimer type.

Hu Q, Cool BH, Wang B, Hearn MG, Martin GM. Hu Q, et al. Among authors: martin gm. Hum Mol Genet. 2002 Feb 15;11(4):465-75. doi: 10.1093/hmg/11.4.465. Hum Mol Genet. 2002. PMID: 11854179

3

Cytotoxicity mediated by conditional expression of a carboxyl-terminal derivative of the beta-amyloid precursor protein.

Sopher BL, Fukuchi K, Smith AC, Leppig KA, Furlong CE, Martin GM. Sopher BL, et al. Among authors: martin gm. Brain Res Mol Brain Res. 1994 Oct;26(1-2):207-17. doi: 10.1016/0169-328x(94)90092-2. Brain Res Mol Brain Res. 1994. PMID: 7854049

4

Selective neurotoxicity of COOH-terminal fragments of the beta-amyloid precursor protein.

Fukuchi K, Sopher B, Furlong CE, Smith AC, Dang N, Martin GM. Fukuchi K, et al. Among authors: martin gm. Neurosci Lett. 1993 May 14;154(1-2):145-8. doi: 10.1016/0304-3940(93)90192-n. Neurosci Lett. 1993. PMID: 8361630

5

Neurodegenerative mechanisms in Alzheimer disease. A role for oxidative damage in amyloid beta protein precursor-mediated cell death.

Sopher BL, Fukuchi K, Kavanagh TJ, Furlong CE, Martin GM. Sopher BL, et al. Among authors: martin gm. Mol Chem Neuropathol. 1996 Oct-Dec;29(2-3):153-68. doi: 10.1007/BF02814999. Mol Chem Neuropathol. 1996. PMID: 8971693

6

The human FE65 gene: genomic structure and an intronic biallelic polymorphism associated with sporadic dementia of the Alzheimer type.

Hu Q, Kukull WA, Bressler SL, Gray MD, Cam JA, Larson EB, Martin GM, Deeb SS. Hu Q, et al. Among authors: martin gm. Hum Genet. 1998 Sep;103(3):295-303. doi: 10.1007/s004390050820. Hum Genet. 1998. PMID: 9799084

7

Alternatively spliced isoforms of FE65 serve as neuron-specific and non-neuronal markers.

Hu Q, Hearn MG, Jin LW, Bressler SL, Martin GM. Hu Q, et al. Among authors: martin gm. J Neurosci Res. 1999 Dec 1;58(5):632-40. doi: 10.1002/(sici)1097-4547(19991201)58:5<632::aid-jnr4>3.0.co;2-p. J Neurosci Res. 1999. PMID: 10561691

8

Isoform-specific knockout of FE65 leads to impaired learning and memory.

Wang B, Hu Q, Hearn MG, Shimizu K, Ware CB, Liggitt DH, Jin LW, Cool BH, Storm DR, Martin GM. Wang B, et al. Among authors: martin gm. J Neurosci Res. 2004 Jan 1;75(1):12-24. doi: 10.1002/jnr.10834. J Neurosci Res. 2004. PMID: 14689444

9

Endoproteolytic cleavage of FE65 converts the adaptor protein to a potent suppressor of the sAPPalpha pathway in primates.

Hu Q, Wang L, Yang Z, Cool BH, Zitnik G, Martin GM. Hu Q, et al. Among authors: martin gm. J Biol Chem. 2005 Apr 1;280(13):12548-58. doi: 10.1074/jbc.M411855200. Epub 2005 Jan 12. J Biol Chem. 2005. PMID: 15647266 Free article.

10

A dominant role for FE65 (APBB1) in nuclear signaling.

Yang Z, Cool BH, Martin GM, Hu Q. Yang Z, et al. Among authors: martin gm. J Biol Chem. 2006 Feb 17;281(7):4207-14. doi: 10.1074/jbc.M508445200. Epub 2005 Dec 6. J Biol Chem. 2006. PMID: 16332686 Free article.

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