Baltimore D - Search Results

1

Regulation of 3' IgH enhancers by a common set of factors, including kappa B-binding proteins.

Michaelson JS, Singh M, Snapper CM, Sha WC, Baltimore D, Birshtein BK. Michaelson JS, et al. Among authors: baltimore d. J Immunol. 1996 Apr 15;156(8):2828-39. J Immunol. 1996. PMID: 8609402

2

B cells from p50/NF-kappa B knockout mice have selective defects in proliferation, differentiation, germ-line CH transcription, and Ig class switching.

Snapper CM, Zelazowski P, Rosas FR, Kehry MR, Tian M, Baltimore D, Sha WC. Snapper CM, et al. Among authors: baltimore d. J Immunol. 1996 Jan 1;156(1):183-91. J Immunol. 1996. PMID: 8598461

3

The p65 subunit of NF-kappa B is redundant with p50 during B cell proliferative responses, and is required for germline CH transcription and class switching to IgG3.

Horwitz BH, Zelazowski P, Shen Y, Wolcott KM, Scott ML, Baltimore D, Snapper CM. Horwitz BH, et al. Among authors: baltimore d. J Immunol. 1999 Feb 15;162(4):1941-6. J Immunol. 1999. PMID: 9973462

4

Constitutive NF-kappa B activation, enhanced granulopoiesis, and neonatal lethality in I kappa B alpha-deficient mice.

Beg AA, Sha WC, Bronson RT, Baltimore D. Beg AA, et al. Among authors: baltimore d. Genes Dev. 1995 Nov 15;9(22):2736-46. doi: 10.1101/gad.9.22.2736. Genes Dev. 1995. PMID: 7590249 Free article.

5

Embryonic lethality and liver degeneration in mice lacking the RelA component of NF-kappa B.

Beg AA, Sha WC, Bronson RT, Ghosh S, Baltimore D. Beg AA, et al. Among authors: baltimore d. Nature. 1995 Jul 13;376(6536):167-70. doi: 10.1038/376167a0. Nature. 1995. PMID: 7603567

6

Targeted disruption of the p50 subunit of NF-kappa B leads to multifocal defects in immune responses.

Sha WC, Liou HC, Tuomanen EI, Baltimore D. Sha WC, et al. Among authors: baltimore d. Cell. 1995 Jan 27;80(2):321-30. doi: 10.1016/0092-8674(95)90415-8. Cell. 1995. PMID: 7834752 Free article.

7

Sequential induction of NF-kappa B/Rel family proteins during B-cell terminal differentiation.

Liou HC, Sha WC, Scott ML, Baltimore D. Liou HC, et al. Among authors: baltimore d. Mol Cell Biol. 1994 Aug;14(8):5349-59. doi: 10.1128/mcb.14.8.5349-5359.1994. Mol Cell Biol. 1994. PMID: 8035813 Free PMC article.

8

Both multiorgan inflammation and myeloid hyperplasia in RelB-deficient mice are T cell dependent.

Weih F, Durham SK, Barton DS, Sha WC, Baltimore D, Bravo R. Weih F, et al. Among authors: baltimore d. J Immunol. 1996 Nov 1;157(9):3974-9. J Immunol. 1996. PMID: 8892630

9

p50-NF-kappaB complexes partially compensate for the absence of RelB: severely increased pathology in p50(-/-)relB(-/-) double-knockout mice.

Weih F, Durham SK, Barton DS, Sha WC, Baltimore D, Bravo R. Weih F, et al. Among authors: baltimore d. J Exp Med. 1997 Apr 7;185(7):1359-70. doi: 10.1084/jem.185.7.1359. J Exp Med. 1997. PMID: 9104822 Free PMC article.

10

The combined absence of NF-kappa B1 and c-Rel reveals that overlapping roles for these transcription factors in the B cell lineage are restricted to the activation and function of mature cells.

Pohl T, Gugasyan R, Grumont RJ, Strasser A, Metcalf D, Tarlinton D, Sha W, Baltimore D, Gerondakis S. Pohl T, et al. Among authors: baltimore d. Proc Natl Acad Sci U S A. 2002 Apr 2;99(7):4514-9. doi: 10.1073/pnas.072071599. Proc Natl Acad Sci U S A. 2002. PMID: 11930006 Free PMC article.

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