Hengstschläger-Ottnad E - Search Results

1

Loss of the p16/MTS1 tumor suppressor gene causes E2F-mediated deregulation of essential enzymes of the DNA precursor metabolism.

Hengstschläger M, Pusch O, Hengstschläger-Ottnad E, Ambros PF, Bernaschek G, Wawra E. Hengstschläger M, et al. DNA Cell Biol. 1996 Jan;15(1):41-51. doi: 10.1089/dna.1996.15.41. DNA Cell Biol. 1996. PMID: 8561896

2

Expression of the cyclin-dependent kinase inhibitor p16 during the ongoing cell cycle.

Soucek T, Pusch O, Hengstschläger-Ottnad E, Wawra E, Bernaschek G, Hengstschläger M. Soucek T, et al. FEBS Lett. 1995 Oct 9;373(2):164-9. doi: 10.1016/0014-5793(95)01033-b. FEBS Lett. 1995. PMID: 7589458 Free article.

3

The role of p16 in the E2F-dependent thymidine kinase regulation.

Hengstschläger M, Hengstschläger-Ottnad E, Pusch O, Wawra E. Hengstschläger M, et al. Among authors: hengstschlager ottnad e. Oncogene. 1996 Apr 18;12(8):1635-43. Oncogene. 1996. PMID: 8622883

4

Specific transformation abolishes cyclin D1 fluctuation throughout the cell cycle.

Pusch O, Soucek T, Wawra E, Hengstschläger-Ottnad E, Bernaschek G, Hengstschläger M. Pusch O, et al. Among authors: hengstschlager ottnad e. FEBS Lett. 1996 May 6;385(3):143-8. doi: 10.1016/0014-5793(96)00369-9. FEBS Lett. 1996. PMID: 8647239 Free article.

5

Deregulated expression of E2F-1 induces cyclin A- and E-associated kinase activities independently from cell cycle position.

Soucek T, Pusch O, Hengstschläger-Ottnad E, Adams PD, Hengstschläger M. Soucek T, et al. Among authors: hengstschlager ottnad e. Oncogene. 1997 May 15;14(19):2251-7. doi: 10.1038/sj.onc.1201061. Oncogene. 1997. PMID: 9178900

6

Cellular targets for activation by c-Myc include the DNA metabolism enzyme thymidine kinase.

Pusch O, Soucek T, Hengstschläger-Ottnad E, Bernaschek G, Hengstschläger M. Pusch O, et al. Among authors: hengstschlager ottnad e. DNA Cell Biol. 1997 Jun;16(6):737-47. doi: 10.1089/dna.1997.16.737. DNA Cell Biol. 1997. PMID: 9212167

7

Quality control of centrifugal elutriation for studies of cell cycle regulations.

Hengstschläger M, Pusch O, Soucek T, Hengstschläger-Ottnad E, Bernaschek G. Hengstschläger M, et al. Among authors: hengstschlager ottnad e. Biotechniques. 1997 Aug;23(2):232-4, 236-7. doi: 10.2144/97232bm12. Biotechniques. 1997. PMID: 9266075 Free article. No abstract available.

8

Cyclin-dependent kinases at the G1-S transition of the mammalian cell cycle.

Hengstschläger M, Braun K, Soucek T, Miloloza A, Hengstschläger-Ottnad E. Hengstschläger M, et al. Among authors: hengstschlager ottnad e. Mutat Res. 1999 Jan;436(1):1-9. doi: 10.1016/s1383-5742(98)00022-2. Mutat Res. 1999. PMID: 9878675 Review.

9

Different regulation of c-Myc- and E2F-1-induced apoptosis during the ongoing cell cycle.

Hengstschläger M, Hölzl G, Hengstschläger-Ottnad E. Hengstschläger M, et al. Among authors: hengstschlager ottnad e. Oncogene. 1999 Jan 21;18(3):843-8. doi: 10.1038/sj.onc.1202342. Oncogene. 1999. PMID: 9989838

10

Tuberous sclerosis gene products in proliferation control.

Hengstschläger M, Rodman DM, Miloloza A, Hengstschläger-Ottnad E, Rosner M, Kubista M. Hengstschläger M, et al. Among authors: hengstschlager ottnad e. Mutat Res. 2001 Jul;488(3):233-9. doi: 10.1016/s1383-5742(01)00058-8. Mutat Res. 2001. PMID: 11397651 Review.

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