App H - Search Results

1

H-ras and raf-1 cooperate in transformation of NIH3T3 fibroblasts.

Cuadrado A, Bruder JT, Heidaran MA, App H, Rapp UR, Aaronson SA. Cuadrado A, et al. Among authors: app h. Oncogene. 1993 Sep;8(9):2443-8. Oncogene. 1993. PMID: 8361757

2

Ras controls coupling of growth factor receptors and protein kinase C in the membrane to Raf-1 and B-Raf protein serine kinases in the cytosol.

Troppmair J, Bruder JT, App H, Cai H, Liptak L, Szeberényi J, Cooper GM, Rapp UR. Troppmair J, et al. Among authors: app h. Oncogene. 1992 Sep;7(9):1867-73. Oncogene. 1992. PMID: 1386920

3

Colony stimulating factor-1 (CSF-1) stimulates temperature dependent phosphorylation and activation of the RAF-1 proto-oncogene product.

Baccarini M, Sabatini DM, App H, Rapp UR, Stanley ER. Baccarini M, et al. Among authors: app h. EMBO J. 1990 Nov;9(11):3649-57. doi: 10.1002/j.1460-2075.1990.tb07576.x. EMBO J. 1990. PMID: 1698619 Free PMC article.

4

Epidermal growth factor (EGF) stimulates association and kinase activity of Raf-1 with the EGF receptor.

App H, Hazan R, Zilberstein A, Ullrich A, Schlessinger J, Rapp U. App H, et al. Mol Cell Biol. 1991 Feb;11(2):913-9. doi: 10.1128/mcb.11.2.913-919.1991. Mol Cell Biol. 1991. PMID: 1990291 Free PMC article.

5

Insulin activates the Raf-1 protein kinase.

Blackshear PJ, Haupt DM, App H, Rapp UR. Blackshear PJ, et al. Among authors: app h. J Biol Chem. 1990 Jul 25;265(21):12131-4. J Biol Chem. 1990. PMID: 2197271 Free article.

6

Raf-1 activates MAP kinase-kinase.

Kyriakis JM, App H, Zhang XF, Banerjee P, Brautigan DL, Rapp UR, Avruch J. Kyriakis JM, et al. Among authors: app h. Nature. 1992 Jul 30;358(6385):417-21. doi: 10.1038/358417a0. Nature. 1992. PMID: 1322500

7

Interleukin 2 induces tyrosine phosphorylation and activation of p72-74 Raf-1 kinase in a T-cell line.

Turner B, Rapp U, App H, Greene M, Dobashi K, Reed J. Turner B, et al. Among authors: app h. Proc Natl Acad Sci U S A. 1991 Feb 15;88(4):1227-31. doi: 10.1073/pnas.88.4.1227. Proc Natl Acad Sci U S A. 1991. PMID: 1996324 Free PMC article.

8

SU5416 is a potent and selective inhibitor of the vascular endothelial growth factor receptor (Flk-1/KDR) that inhibits tyrosine kinase catalysis, tumor vascularization, and growth of multiple tumor types.

Fong TA, Shawver LK, Sun L, Tang C, App H, Powell TJ, Kim YH, Schreck R, Wang X, Risau W, Ullrich A, Hirth KP, McMahon G. Fong TA, et al. Among authors: app h. Cancer Res. 1999 Jan 1;59(1):99-106. Cancer Res. 1999. PMID: 9892193

9

Flk-1 as a target for tumor growth inhibition.

Strawn LM, McMahon G, App H, Schreck R, Kuchler WR, Longhi MP, Hui TH, Tang C, Levitzki A, Gazit A, Chen I, Keri G, Orfi L, Risau W, Flamme I, Ullrich A, Hirth KP, Shawver LK. Strawn LM, et al. Among authors: app h. Cancer Res. 1996 Aug 1;56(15):3540-5. Cancer Res. 1996. PMID: 8758924

10

A tumor-derived protein which provides T-cell costimulation through accessory cell activation.

Powell TJ, Schreck R, McCall M, Hui T, Rice A, App H, Azam M, Ullrich A, Shawver LK. Powell TJ, et al. Among authors: app h. J Immunother Emphasis Tumor Immunol. 1995 May;17(4):209-21. doi: 10.1097/00002371-199505000-00003. J Immunother Emphasis Tumor Immunol. 1995. PMID: 7582257

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