Ayer DE - Search Results

1

Mapping of two genes encoding members of a distinct subfamily of MAX interacting proteins: MAD to human chromosome 2 and mouse chromosome 6, and MXI1 to human chromosome 10 and mouse chromosome 19.

Edelhoff S, Ayer DE, Zervos AS, Steingrímsson E, Jenkins NA, Copeland NG, Eisenman RN, Brent R, Disteche CM. Edelhoff S, et al. Among authors: ayer de. Oncogene. 1994 Feb;9(2):665-8. Oncogene. 1994. PMID: 8290278

2

The Max transcription factor network: involvement of Mad in differentiation and an approach to identification of target genes.

Hurlin PJ, Ayer DE, Grandori C, Eisenman RN. Hurlin PJ, et al. Among authors: ayer de. Cold Spring Harb Symp Quant Biol. 1994;59:109-16. doi: 10.1101/sqb.1994.059.01.014. Cold Spring Harb Symp Quant Biol. 1994. PMID: 7587059 Review.

3

Mad-Max transcriptional repression is mediated by ternary complex formation with mammalian homologs of yeast repressor Sin3.

Ayer DE, Lawrence QA, Eisenman RN. Ayer DE, et al. Cell. 1995 Mar 10;80(5):767-76. doi: 10.1016/0092-8674(95)90355-0. Cell. 1995. PMID: 7889570 Free article.

4

A switch from Myc:Max to Mad:Max heterocomplexes accompanies monocyte/macrophage differentiation.

Ayer DE, Eisenman RN. Ayer DE, et al. Genes Dev. 1993 Nov;7(11):2110-9. doi: 10.1101/gad.7.11.2110. Genes Dev. 1993. PMID: 8224841 Free article.

5

Mad: a heterodimeric partner for Max that antagonizes Myc transcriptional activity.

Ayer DE, Kretzner L, Eisenman RN. Ayer DE, et al. Cell. 1993 Jan 29;72(2):211-22. doi: 10.1016/0092-8674(93)90661-9. Cell. 1993. PMID: 8425218

6

Mad3 and Mad4: novel Max-interacting transcriptional repressors that suppress c-myc dependent transformation and are expressed during neural and epidermal differentiation.

Hurlin PJ, Quéva C, Koskinen PJ, Steingrímsson E, Ayer DE, Copeland NG, Jenkins NA, Eisenman RN. Hurlin PJ, et al. Among authors: ayer de. EMBO J. 1995 Nov 15;14(22):5646-59. doi: 10.1002/j.1460-2075.1995.tb00252.x. EMBO J. 1995. PMID: 8521822 Free PMC article.

7

Regulation of Myc and Mad during epidermal differentiation and HPV-associated tumorigenesis.

Hurlin PJ, Foley KP, Ayer DE, Eisenman RN, Hanahan D, Arbeit JM. Hurlin PJ, et al. Among authors: ayer de. Oncogene. 1995 Dec 21;11(12):2487-501. Oncogene. 1995. PMID: 8545105

8

Mad proteins contain a dominant transcription repression domain.

Ayer DE, Laherty CD, Lawrence QA, Armstrong AP, Eisenman RN. Ayer DE, et al. Mol Cell Biol. 1996 Oct;16(10):5772-81. doi: 10.1128/MCB.16.10.5772. Mol Cell Biol. 1996. PMID: 8816491 Free PMC article.

9

Repression of Myc-Ras cotransformation by Mad is mediated by multiple protein-protein interactions.

Koskinen PJ, Ayer DE, Eisenman RN. Koskinen PJ, et al. Among authors: ayer de. Cell Growth Differ. 1995 Jun;6(6):623-9. Cell Growth Differ. 1995. PMID: 7669717

10

Myc-Max heterodimers activate a DEAD box gene and interact with multiple E box-related sites in vivo.

Grandori C, Mac J, Siëbelt F, Ayer DE, Eisenman RN. Grandori C, et al. Among authors: ayer de. EMBO J. 1996 Aug 15;15(16):4344-57. EMBO J. 1996. PMID: 8861962 Free PMC article.

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