Hattori M - Search Results

1

Protection against adoptive transfer of autoimmune diabetes mediated through very late antigen-4 integrin.

Burkly LC, Jakubowski A, Hattori M. Burkly LC, et al. Among authors: hattori m. Diabetes. 1994 Apr;43(4):529-34. doi: 10.2337/diab.43.4.529. Diabetes. 1994. PMID: 8138057

2

Fetal or neonatal low-glycotoxin environment prevents autoimmune diabetes in NOD mice.

Peppa M, He C, Hattori M, McEvoy R, Zheng F, Vlassara H. Peppa M, et al. Among authors: hattori m. Diabetes. 2003 Jun;52(6):1441-8. doi: 10.2337/diabetes.52.6.1441. Diabetes. 2003. PMID: 12765955

3

Reactive oxygen intermediates in autoimmune islet cell destruction of the NOD mouse induced by peritoneal exudate cells (rich in macrophages) but not T cells.

Horio F, Fukuda M, Katoh H, Petruzzelli M, Yano N, Rittershaus C, Bonner-Weir S, Hattori M. Horio F, et al. Among authors: hattori m. Diabetologia. 1994 Jan;37(1):22-31. doi: 10.1007/BF00428773. Diabetologia. 1994. PMID: 8150225

4

Radioassay determination of insulin autoantibodies in NOD mice. Correlation with increased risk of progression to overt diabetes.

Ziegler AG, Vardi P, Ricker AT, Hattori M, Soeldner JS, Eisenbarth GS. Ziegler AG, et al. Among authors: hattori m. Diabetes. 1989 Mar;38(3):358-63. doi: 10.2337/diab.38.3.358. Diabetes. 1989. PMID: 2917700

5

Analysis of the T cell receptor (TcR) regions in the NOD, NON and CTS mouse strains define new TcR V alpha haplotypes and new deletions in the TcR V beta region.

Lund T, Shaikh S, Hattori M, Makino S. Lund T, et al. Among authors: hattori m. Eur J Immunol. 1992 Mar;22(3):871-4. doi: 10.1002/eji.1830220337. Eur J Immunol. 1992. PMID: 1532150

6

IGF-1 decreases collagen degradation in diabetic NOD mesangial cells: implications for diabetic nephropathy.

Lupia E, Elliot SJ, Lenz O, Zheng F, Hattori M, Striker GE, Striker LJ. Lupia E, et al. Among authors: hattori m. Diabetes. 1999 Aug;48(8):1638-44. doi: 10.2337/diabetes.48.8.1638. Diabetes. 1999. PMID: 10426384

We showed that mesangial cells (MCs) from diabetic mice exhibited a stable phenotypic switch, consisting of both increased IGF-1 synthesis and proliferation (Elliot SJ, Striker LJ, Hattori M, Yang CW, He CJ, Peten EP, Striker GE: Mesangial cells from diabetic NOD mi …

We showed that mesangial cells (MCs) from diabetic mice exhibited a stable phenotypic switch, consisting of both increased IGF-1 synthesis a …

7

Physiological beta cell death triggers priming of self-reactive T cells by dendritic cells in a type-1 diabetes model.

Turley S, Poirot L, Hattori M, Benoist C, Mathis D. Turley S, et al. Among authors: hattori m. J Exp Med. 2003 Nov 17;198(10):1527-37. doi: 10.1084/jem.20030966. J Exp Med. 2003. PMID: 14623908 Free PMC article.

8

Cutting edge: homologous recombination of the MHC class I K region defines new MHC-linked diabetogenic susceptibility gene(s) in nonobese diabetic mice.

Hattori M, Yamato E, Itoh N, Senpuku H, Fujisawa T, Yoshino M, Fukuda M, Matsumoto E, Toyonaga T, Nakagawa I, Petruzzelli M, McMurray A, Weiner H, Sagai T, Moriwaki K, Shiroishi T, Maron R, Lund T. Hattori M, et al. J Immunol. 1999 Aug 15;163(4):1721-4. J Immunol. 1999. PMID: 10438898

9

RFLP analysis of the MHC class III region defines unique haplotypes for the non-obese diabetic, cataract Shionogi and the non-obese non-diabetic mouse strains.

Lund T, Shaikh S, Kendall E, Campbell RD, Hattori M, Makino S, Cooke A. Lund T, et al. Among authors: hattori m. Diabetologia. 1993 Aug;36(8):727-33. doi: 10.1007/BF00401143. Diabetologia. 1993. PMID: 8104833

10

Major histocompatibility complex-linked diabetogenic gene of the nonobese diabetic mouse. Analysis of genomic DNA amplified by the polymerase chain reaction.

Ikegami H, Eisenbarth GS, Hattori M. Ikegami H, et al. Among authors: hattori m. J Clin Invest. 1990 Jan;85(1):18-24. doi: 10.1172/JCI114410. J Clin Invest. 1990. PMID: 2295694 Free PMC article.

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