Kishi M - Search Results

1

High susceptibility of U937-derived subclones to infection with human immunodeficiency virus type 1 is correlated with virus-induced cell differentiation and superoxide generation.

Kameoka M, Kimura T, Okada Y, Nakaya T, Kishi M, Ikuta K. Kameoka M, et al. Among authors: kishi m. Immunopharmacology. 1995 Jun;30(1):89-101. doi: 10.1016/0162-3109(95)00012-i. Immunopharmacology. 1995. PMID: 7591717

2

Viral activation from latency during retrodifferentiation of U937 cells exposed to phorbol ester followed by infection with human immunodeficiency virus type 1.

Okada Y, Kimura T, Kameoka M, Kishi M, Azuma I, Ikuta K. Okada Y, et al. Among authors: kishi m. Immunopharmacology. 1995 Jun;30(1):27-39. doi: 10.1016/0162-3109(95)98646-k. Immunopharmacology. 1995. PMID: 7591711

3

High susceptibility of U937-derived subclones to human immunodeficiency virus type 1 infection correlates with accumulation of unintegrated circular viral DNA.

Kameoka M, Kimura T, Okada Y, Fujinaga K, Nakaya T, Takahashi H, Kishi M, Ikuta K. Kameoka M, et al. Among authors: kishi m. Virus Genes. 1996;12(2):117-29. doi: 10.1007/BF00572950. Virus Genes. 1996. PMID: 8879128

4

Human immunodeficiency virus type 1 (HIV-1) superinfection of a cell clone converting it from production of defective to infectious HIV-1 is mediated predominantly by CD4 regions other than the major binding site for HIV-1 glycoproteins.

Ohki K, Kishi M, Ohmura K, Morikawa Y, Jones IM, Azuma I, Ikuta K. Ohki K, et al. Among authors: kishi m. J Gen Virol. 1992 Jul;73 ( Pt 7):1761-72. doi: 10.1099/0022-1317-73-7-1761. J Gen Virol. 1992. PMID: 1629701

5

Cells surviving infection by human immunodeficiency virus type 1: vif or vpu mutants produce non-infectious or markedly less cytopathic viruses.

Kishi M, Nishino Y, Sumiya M, Ohki K, Kimura T, Goto T, Nakai M, Kakinuma M, Ikuta K. Kishi M, et al. J Gen Virol. 1992 Jan;73 ( Pt 1):77-87. doi: 10.1099/0022-1317-73-1-77. J Gen Virol. 1992. PMID: 1730943

6

Human immunodeficiency virus type 1 vif, vpr, and vpu mutants can produce persistently infected cells.

Nishino Y, Kishi M, Sumiya M, Ogawa K, Adachi A, Maotani-Imai K, Kato S, Hirai K, Ikuta K. Nishino Y, et al. Among authors: kishi m. Arch Virol. 1991;120(3-4):181-92. doi: 10.1007/BF01310474. Arch Virol. 1991. PMID: 1835572

7

Naturally occurring accessory gene mutations lead to persistent human immunodeficiency virus type 1 infection of CD4-positive T cells.

Kishi M, Zheng YH, Bahmani MK, Tokunaga K, Takahashi H, Kakinuma M, Lai PK, Nonoyama M, Luftig RB, Ikuta K. Kishi M, et al. J Virol. 1995 Dec;69(12):7507-18. doi: 10.1128/JVI.69.12.7507-7518.1995. J Virol. 1995. PMID: 7494257 Free PMC article.

8

Persistent infection of MT-4 cells by human immunodeficiency virus type 1 becomes increasingly likely with in vitro serial passage of wild-type but not nef mutant virus.

Nishino Y, Nakaya T, Fujinaga K, Kishi M, Azuma I, Ikuta K. Nishino Y, et al. Among authors: kishi m. J Gen Virol. 1994 Sep;75 ( Pt 9):2241-51. doi: 10.1099/0022-1317-75-9-2241. J Gen Virol. 1994. PMID: 7521392

9

Demonstration of human Borna disease virus RNA in human peripheral blood mononuclear cells.

Kishi M, Nakaya T, Nakamura Y, Zhong Q, Ikeda K, Senjo M, Kakinuma M, Kato S, Ikuta K. Kishi M, et al. FEBS Lett. 1995 May 15;364(3):293-7. doi: 10.1016/0014-5793(95)00406-y. FEBS Lett. 1995. PMID: 7538936 Free article.

10

Nonsense mutations in the vpr gene of HIV-1 during in vitro virus passage and in HIV-1 carrier-derived peripheral blood mononuclear cells.

Nakaya T, Fujinaga K, Kishi M, Oka S, Kurata T, Jones IM, Ikuta K. Nakaya T, et al. Among authors: kishi m. FEBS Lett. 1994 Oct 31;354(1):17-22. doi: 10.1016/0014-5793(94)01074-9. FEBS Lett. 1994. PMID: 7957894 Free article.

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