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Novel mechanisms of fibroblast growth factor 1 function.
Maciag T, Zhan X, Garfinkel S, Friedman S, Prudovsky I, Jackson A, Wessendorf J, Hu X, Gamble S, Shi J, et al. Maciag T, et al. Recent Prog Horm Res. 1994;49:105-23. doi: 10.1016/b978-0-12-571149-4.50009-x. Recent Prog Horm Res. 1994. PMID: 7511824 Review. No abstract available.
The cysteine residue responsible for the release of fibroblast growth factor-1 residues in a domain independent of the domain for phosphatidylserine binding.
Tarantini F, Gamble S, Jackson A, Maciag T. Tarantini F, et al. Among authors: maciag t. J Biol Chem. 1995 Dec 8;270(49):29039-42. doi: 10.1074/jbc.270.49.29039. J Biol Chem. 1995. PMID: 7493920 Free article.
We have previously reported that FGF-1 is released in response to temperature stress as a latent homodimer through a pathway that is potentiated by the Golgi inhibitor, brefeldin A (Jackson, A., Tarantini, F., Gamble, S., Friedman, S., and Maciag, T. (1995) J. Biol. …
We have previously reported that FGF-1 is released in response to temperature stress as a latent homodimer through a pathway that is potenti …
Intact and functional fibroblast growth factor (FGF) receptor-1 trafficks near the nucleus in response to FGF-1.
Prudovsky I, Savion N, Zhan X, Friesel R, Xu J, Hou J, McKeehan WL, Maciag T. Prudovsky I, et al. Among authors: maciag t. J Biol Chem. 1994 Dec 16;269(50):31720-4. J Biol Chem. 1994. PMID: 7527394 Free article.
Exogenous fibroblast growth factor-1 (FGF-1) associates with the nucleus in a receptor-dependent manner during the entire G1 period of the BALB/c 3T3 cell cycle (Zhan, X., Hu, X., Friesel, R., and Maciag, T. (1993) J. Biol. Chem. 268, 9611-9620). To further study th …
Exogenous fibroblast growth factor-1 (FGF-1) associates with the nucleus in a receptor-dependent manner during the entire G1 period of the B …
The release of fibroblast growth factor-1 from NIH 3T3 cells in response to temperature involves the function of cysteine residues.
Jackson A, Tarantini F, Gamble S, Friedman S, Maciag T. Jackson A, et al. Among authors: maciag t. J Biol Chem. 1995 Jan 6;270(1):33-6. doi: 10.1074/jbc.270.1.33. J Biol Chem. 1995. PMID: 7529229 Free article.
Fibroblast growth factor (FGF)-1 is released from NIH 3T3 cells in response to heat shock as a biologically inactive protein that is unable to bind heparin and requires activation by (NH4)2SO4 to generate a biologically active extracellular heparin-binding growth factor (Jackson, …
Fibroblast growth factor (FGF)-1 is released from NIH 3T3 cells in response to heat shock as a biologically inactive protein that is unable …
FGF-1-dependent proliferative and migratory responses are impaired in senescent human umbilical vein endothelial cells and correlate with the inability to signal tyrosine phosphorylation of fibroblast growth factor receptor-1 substrates.
Garfinkel S, Hu X, Prudovsky IA, McMahon GA, Kapnik EM, McDowell SD, Maciag T. Garfinkel S, et al. Among authors: maciag t. J Cell Biol. 1996 Aug;134(3):783-91. doi: 10.1083/jcb.134.3.783. J Cell Biol. 1996. PMID: 8707855 Free PMC article.
155 results