Groveman BR - Search Results

1

Limbic system synaptic dysfunctions associated with prion disease onset.

Foliaki ST, Groveman BR, Dews EA, Williams K, El Soufi H, Schwarz B, Leung JM, Schneider CA, Schwartz CL, Bohrnsen E, Kimzey CD, Race B, Haigh CL. Foliaki ST, et al. Among authors: groveman br. Acta Neuropathol Commun. 2024 Dec 20;12(1):192. doi: 10.1186/s40478-024-01905-w. Acta Neuropathol Commun. 2024. PMID: 39707496 Free PMC article.

2

Sporadic Creutzfeldt-Jakob disease prion infection of human cerebral organoids.

Groveman BR, Foliaki ST, Orru CD, Zanusso G, Carroll JA, Race B, Haigh CL. Groveman BR, et al. Acta Neuropathol Commun. 2019 Jun 14;7(1):90. doi: 10.1186/s40478-019-0742-2. Acta Neuropathol Commun. 2019. PMID: 31196223 Free PMC article.

3

Cerebral organoids as a new model for prion disease.

Groveman BR, Smith A, Williams K, Haigh CL. Groveman BR, et al. PLoS Pathog. 2021 Jul 21;17(7):e1009747. doi: 10.1371/journal.ppat.1009747. eCollection 2021 Jul. PLoS Pathog. 2021. PMID: 34288977 Free PMC article. Review. No abstract available.

4

Reduced SOD2 expression does not influence prion disease course or pathology in mice.

Foliaki ST, Race B, Williams K, Baune C, Groveman BR, Haigh CL. Foliaki ST, et al. Among authors: groveman br. PLoS One. 2021 Nov 4;16(11):e0259597. doi: 10.1371/journal.pone.0259597. eCollection 2021. PLoS One. 2021. PMID: 34735539 Free PMC article.

5

Sporadic Creutzfeldt-Jakob disease infected human cerebral organoids retain the original human brain subtype features following transmission to humanized transgenic mice.

Groveman BR, Race B, Foliaki ST, Williams K, Hughson AG, Baune C, Zanusso G, Haigh CL. Groveman BR, et al. Acta Neuropathol Commun. 2023 Feb 14;11(1):28. doi: 10.1186/s40478-023-01512-1. Acta Neuropathol Commun. 2023. PMID: 36788566 Free PMC article.

6

Temporary alteration of neuronal network communication is a protective response to redox imbalance that requires GPI-anchored prion protein.

Foliaki ST, Wood A, Williams K, Smith A, Walters RO, Baune C, Groveman BR, Haigh CL. Foliaki ST, et al. Among authors: groveman br. Redox Biol. 2023 Jul;63:102733. doi: 10.1016/j.redox.2023.102733. Epub 2023 May 5. Redox Biol. 2023. PMID: 37172395 Free PMC article.

7

Using our mini-brains: cerebral organoids as an improved cellular model for human prion disease.

Groveman BR, Walters R, Haigh CL. Groveman BR, et al. Neural Regen Res. 2020 Jun;15(6):1019-1020. doi: 10.4103/1673-5374.270300. Neural Regen Res. 2020. PMID: 31823872 Free PMC article. Review. No abstract available.

8

Prion protein N1 cleavage peptides stimulate microglial interaction with surrounding cells.

Carroll JA, Groveman BR, Williams K, Moore R, Race B, Haigh CL. Carroll JA, et al. Among authors: groveman br. Sci Rep. 2020 Apr 20;10(1):6654. doi: 10.1038/s41598-020-63472-z. Sci Rep. 2020. PMID: 32313035 Free PMC article.

9

Pathogenic Prion Protein Isoforms Are Not Present in Cerebral Organoids Generated from Asymptomatic Donors Carrying the E200K Mutation Associated with Familial Prion Disease.

Foliaki ST, Groveman BR, Yuan J, Walters R, Zhang S, Tesar P, Zou W, Haigh CL. Foliaki ST, et al. Among authors: groveman br. Pathogens. 2020 Jun 18;9(6):482. doi: 10.3390/pathogens9060482. Pathogens. 2020. PMID: 32570796 Free PMC article.

10

Neuronal excitatory-to-inhibitory balance is altered in cerebral organoid models of genetic neurological diseases.

Foliaki ST, Schwarz B, Groveman BR, Walters RO, Ferreira NC, Orrù CD, Smith A, Wood A, Schmit OM, Freitag P, Yuan J, Zou W, Bosio CM, Carroll JA, Haigh CL. Foliaki ST, et al. Among authors: groveman br. Mol Brain. 2021 Oct 11;14(1):156. doi: 10.1186/s13041-021-00864-w. Mol Brain. 2021. PMID: 34635127 Free PMC article.

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