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Sema4D/PlexinB1 inhibition ameliorates blood-brain barrier damage and improves outcome after stroke in rats.
Zhou YF, Li YN, Jin HJ, Wu JH, He QW, Wang XX, Lei H, Hu B. Zhou YF, et al. FASEB J. 2018 Apr;32(4):2181-2196. doi: 10.1096/fj.201700786RR. Epub 2018 Jan 5. FASEB J. 2018. PMID: 29242274
Our study indicates that Sema4D may be a novel therapeutic target for treatment in the acute phase of stroke.-Zhou, Y.-F., Li, Y.-N., Jin, H.-J., Wu, J.-H., He, Q.-W., Wang, X.-X., Lei, H., Hu, B. ...
Our study indicates that Sema4D may be a novel therapeutic target for treatment in the acute phase of stroke.-Zhou, Y.-F., Li, …
Semaphorin-3A protects against neointimal hyperplasia after vascular injury.
Wu JH, Zhou YF, Hong CD, Chen AQ, Luo Y, Mao L, Xia YP, He QW, Jin HJ, Huang M, Li YN, Hu B. Wu JH, et al. Among authors: zhou yf. EBioMedicine. 2019 Jan;39:95-108. doi: 10.1016/j.ebiom.2018.12.023. Epub 2018 Dec 19. EBioMedicine. 2019. PMID: 30579864 Free PMC article.
Sema3E/PlexinD1 signaling inhibits postischemic angiogenesis by regulating endothelial DLL4 and filopodia formation in a rat model of ischemic stroke.
Zhou YF, Chen AQ, Wu JH, Mao L, Xia YP, Jin HJ, He QW, Miao QR, Yue ZY, Liu XL, Huang M, Li YN, Hu B. Zhou YF, et al. FASEB J. 2019 Apr;33(4):4947-4961. doi: 10.1096/fj.201801706RR. Epub 2019 Jan 17. FASEB J. 2019. PMID: 30653356
Our study reveals that Sema3E/PlexinD1 signaling, which suppressed endothelial DLL4 expression, cell motility, and filopodia formation, is expected to be a novel druggable target for angiogenesis during poststroke progression.-Zhou, Y.-F., Chen, A.-Q., Wu, J.-H., Ma …
Our study reveals that Sema3E/PlexinD1 signaling, which suppressed endothelial DLL4 expression, cell motility, and filopodia formation, is e …
73,910 results
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