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Docking and stability defects in mitofusin highlight the proteasome as a potential therapeutic target.
iScience. 2023 Jun 7;26(7):107014. doi: 10.1016/j.isci.2023.107014. eCollection 2023 Jul 21.
iScience. 2023.
PMID: 37416455
Free PMC article.
Plasticity in salt bridge allows fusion-competent ubiquitylation of mitofusins and Cdc48 recognition.
Anton V, Buntenbroich I, Schuster R, Babatz F, Simões T, Altin S, Calabrese G, Riemer J, Schauss A, Escobar-Henriques M.
Anton V, et al. Among authors: buntenbroich i.
Life Sci Alliance. 2019 Nov 18;2(6):e201900491. doi: 10.26508/lsa.201900491. Print 2019 Dec.
Life Sci Alliance. 2019.
PMID: 31740565
Free PMC article.
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Analysis of Protein Stability by Synthesis Shutoff.
Buntenbroich I, Simões T, Escobar-Henriques M.
Buntenbroich I, et al.
Bio Protoc. 2021 Nov 20;11(22):e4225. doi: 10.21769/BioProtoc.4225. eCollection 2021 Nov 20.
Bio Protoc. 2021.
PMID: 34909446
Free PMC article.
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E4 ubiquitin ligase promotes mitofusin turnover and mitochondrial stress response.
Anton V, Buntenbroich I, Simões T, Joaquim M, Müller L, Buettner R, Odenthal M, Hoppe T, Escobar-Henriques M.
Anton V, et al. Among authors: buntenbroich i.
Mol Cell. 2023 Aug 17;83(16):2976-2990.e9. doi: 10.1016/j.molcel.2023.07.021.
Mol Cell. 2023.
PMID: 37595558
Free PMC article.
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