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Senescence-associated oxidative DNA damage promotes the generation of neoplastic cells.
Gosselin K, Martien S, Pourtier A, Vercamer C, Ostoich P, Morat L, Sabatier L, Duprez L, T'kint de Roodenbeke C, Gilson E, Malaquin N, Wernert N, Slijepcevic P, Ashtari M, Chelli F, Deruy E, Vandenbunder B, De Launoit Y, Abbadie C. Gosselin K, et al. Among authors: abbadie c. Cancer Res. 2009 Oct 15;69(20):7917-25. doi: 10.1158/0008-5472.CAN-08-2510. Epub 2009 Oct 13. Cancer Res. 2009. PMID: 19826058
HIC1 (hypermethylated in cancer 1) SUMOylation is dispensable for DNA repair but is essential for the apoptotic DNA damage response (DDR) to irreparable DNA double-strand breaks (DSBs).
Paget S, Dubuissez M, Dehennaut V, Nassour J, Harmon BT, Spruyt N, Loison I, Abbadie C, Rood BR, Leprince D. Paget S, et al. Among authors: abbadie c. Oncotarget. 2017 Jan 10;8(2):2916-2935. doi: 10.18632/oncotarget.13807. Oncotarget. 2017. PMID: 27935866 Free PMC article.
The out-of-field dose in radiation therapy induces delayed tumorigenesis by senescence evasion.
Goy E, Tomezak M, Facchin C, Martin N, Bouchaert E, Benoit J, de Schutter C, Nassour J, Saas L, Drullion C, Brodin PM, Vandeputte A, Molendi-Coste O, Pineau L, Goormachtigh G, Pluquet O, Pourtier A, Cleri F, Lartigau E, Penel N, Abbadie C. Goy E, et al. Among authors: abbadie c. Elife. 2022 Mar 18;11:e67190. doi: 10.7554/eLife.67190. Elife. 2022. PMID: 35302491 Free PMC article.
113 results