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Intracellular persistence of Staphylococcus aureus in endothelial cells is promoted by the absence of phenol-soluble modulins.
Siegmund A, Afzal MA, Tetzlaff F, Keinhörster D, Gratani F, Paprotka K, Westermann M, Nietzsche S, Wolz C, Fraunholz M, Hübner CA, Löffler B, Tuchscherr L. Siegmund A, et al. Among authors: keinhorster d. Virulence. 2021 Dec;12(1):1186-1198. doi: 10.1080/21505594.2021.1910455. Virulence. 2021. PMID: 33843450 Free PMC article.
Wall teichoic acids mediate increased virulence in Staphylococcus aureus.
Wanner S, Schade J, Keinhörster D, Weller N, George SE, Kull L, Bauer J, Grau T, Winstel V, Stoy H, Kretschmer D, Kolata J, Wolz C, Bröker BM, Weidenmaier C. Wanner S, et al. Among authors: keinhorster d. Nat Microbiol. 2017 Jan 23;2:16257. doi: 10.1038/nmicrobiol.2016.257. Nat Microbiol. 2017. PMID: 28112716
Corrigendum: Wall teichoic acids mediate increased virulence in Staphylococcus aureus.
Wanner S, Schade J, Keinhörster D, Weller N, George SE, Kull L, Bauer J, Grau T, Winstel V, Stoy H, Kretschmer D, Kolata J, Wolz C, Bröker BM, Weidenmaier C. Wanner S, et al. Among authors: keinhorster d. Nat Microbiol. 2017 Mar 13;2:17048. doi: 10.1038/nmicrobiol.2017.48. Nat Microbiol. 2017. PMID: 28287624 No abstract available.