Chahine M - Search Results

1

iPSC-derived cardiomyocytes from patients with myotonic dystrophy type 1 have abnormal ion channel functions and slower conduction velocities.

Poulin H, Mercier A, Djemai M, Pouliot V, Deschenes I, Boutjdir M, Puymirat J, Chahine M. Poulin H, et al. Among authors: chahine m. Sci Rep. 2021 Jan 28;11(1):2500. doi: 10.1038/s41598-021-82007-8. Sci Rep. 2021. PMID: 33510259 Free PMC article.

2

Increase in the proliferative capacity of human myoblasts by using the T antigen under the vimentin promoter control.

Deschênes I, Chahine M, Tremblay J, Paulin D, Puymirat J. Deschênes I, et al. Among authors: chahine m. Muscle Nerve. 1997 Apr;20(4):437-45. doi: 10.1002/(sici)1097-4598(199704)20:4<437::aid-mus6>3.0.co;2-b. Muscle Nerve. 1997. PMID: 9121501

3

Restoration of fast inactivation in an inactivation-defective human heart sodium channel by the cysteine modifying reagent benzyl-MTS: analysis of IFM-ICM mutation.

Chahine M, Deschênes I, Trottier E, Chen LQ, Kallen RG. Chahine M, et al. Biochem Biophys Res Commun. 1997 Apr 28;233(3):606-10. doi: 10.1006/bbrc.1997.6510. Biochem Biophys Res Commun. 1997. PMID: 9168898 Free article.

4

Myotonic dystrophy kinase modulates skeletal muscle but not cardiac voltage-gated sodium channels.

Chahine M, George AL Jr. Chahine M, et al. FEBS Lett. 1997 Aug 4;412(3):621-4. doi: 10.1016/s0014-5793(97)00869-7. FEBS Lett. 1997. PMID: 9276478 Free article.

5

Electrophysiological study of chimeric sodium channels from heart and skeletal muscle.

Deschênes I, Chen L, Kallen RG, Chahine M. Deschênes I, et al. Among authors: chahine m. J Membr Biol. 1998 Jul 1;164(1):25-34. doi: 10.1007/s002329900390. J Membr Biol. 1998. PMID: 9636241

6

Cysteine scanning analysis of the IFM cluster in the inactivation gate of a human heart sodium channel.

Deschênes I, Trottier E, Chahine M. Deschênes I, et al. Among authors: chahine m. Cardiovasc Res. 1999 May;42(2):521-9. doi: 10.1016/s0008-6363(99)00064-4. Cardiovasc Res. 1999. PMID: 10533587

7

SCN5A mutation (T1620M) causing Brugada syndrome exhibits different phenotypes when expressed in Xenopus oocytes and mammalian cells.

Baroudi G, Carbonneau E, Pouliot V, Chahine M. Baroudi G, et al. Among authors: chahine m. FEBS Lett. 2000 Feb 4;467(1):12-6. doi: 10.1016/s0014-5793(00)01099-1. FEBS Lett. 2000. PMID: 10664447 Free article.

8

Electrophysiological characterization of SCN5A mutations causing long QT (E1784K) and Brugada (R1512W and R1432G) syndromes.

Deschênes I, Baroudi G, Berthet M, Barde I, Chalvidan T, Denjoy I, Guicheney P, Chahine M. Deschênes I, et al. Among authors: chahine m. Cardiovasc Res. 2000 Apr;46(1):55-65. doi: 10.1016/s0008-6363(00)00006-7. Cardiovasc Res. 2000. PMID: 10727653

9

Novel mechanism for Brugada syndrome: defective surface localization of an SCN5A mutant (R1432G).

Baroudi G, Pouliot V, Denjoy I, Guicheney P, Shrier A, Chahine M. Baroudi G, et al. Among authors: chahine m. Circ Res. 2001 Jun 22;88(12):E78-83. doi: 10.1161/hh1201.093270. Circ Res. 2001. PMID: 11420310

10

Implication of the C-terminal region of the alpha-subunit of voltage-gated sodium channels in fast inactivation.

Deschênes I, Trottier E, Chahine M. Deschênes I, et al. Among authors: chahine m. J Membr Biol. 2001 Sep 15;183(2):103-14. doi: 10.1007/s00232-001-0058-5. J Membr Biol. 2001. PMID: 11562792

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