Mitchell L - Search Results

1

Deep phenotyping detects a pathological CD4⁺ T-cell complosome signature in systemic sclerosis.

Arbore G, Ong VH, Costantini B, Denton CP, Abraham D, Placais L, Blighe K, Mitchell L, Ellis R, Heck S, Nocerino P, Woodruff TM, Kordasti S, Kemper C, Hourcade DE. Arbore G, et al. Among authors: mitchell l. Cell Mol Immunol. 2020 Sep;17(9):1010-1013. doi: 10.1038/s41423-019-0360-8. Epub 2020 Jan 23. Cell Mol Immunol. 2020. PMID: 31974397 Free PMC article. No abstract available.

2

A conserved element in the serine protease domain of complement factor B.

Hourcade DE, Mitchell LM, Oglesby TJ. Hourcade DE, et al. Among authors: mitchell lm. J Biol Chem. 1998 Oct 2;273(40):25996-6000. doi: 10.1074/jbc.273.40.25996. J Biol Chem. 1998. PMID: 9748277 Free article.

3

Mutations of the type A domain of complement factor B that promote high-affinity C3b-binding.

Hourcade DE, Mitchell LM, Oglesby TJ. Hourcade DE, et al. Among authors: mitchell lm. J Immunol. 1999 Mar 1;162(5):2906-11. J Immunol. 1999. PMID: 10072540

4

Decay acceleration of the complement alternative pathway C3 convertase.

Hourcade DE, Mitchell LM, Medof ME. Hourcade DE, et al. Among authors: mitchell lm. Immunopharmacology. 1999 May;42(1-3):167-73. doi: 10.1016/s0162-3109(99)00005-3. Immunopharmacology. 1999. PMID: 10408377

5

Characterization of the active sites in decay-accelerating factor.

Kuttner-Kondo LA, Mitchell L, Hourcade DE, Medof ME. Kuttner-Kondo LA, et al. Among authors: mitchell l. J Immunol. 2001 Aug 15;167(4):2164-71. doi: 10.4049/jimmunol.167.4.2164. J Immunol. 2001. PMID: 11490001

6

Decay-accelerating factor (DAF), complement receptor 1 (CR1), and factor H dissociate the complement AP C3 convertase (C3bBb) via sites on the type A domain of Bb.

Hourcade DE, Mitchell L, Kuttner-Kondo LA, Atkinson JP, Medof ME. Hourcade DE, et al. Among authors: mitchell l. J Biol Chem. 2002 Jan 11;277(2):1107-12. doi: 10.1074/jbc.M109322200. Epub 2001 Nov 2. J Biol Chem. 2002. PMID: 11694537 Free article.

7

A novel inhibitor of the alternative complement pathway prevents antiphospholipid antibody-induced pregnancy loss in mice.

Thurman JM, Kraus DM, Girardi G, Hourcade D, Kang HJ, Royer PA, Mitchell LM, Giclas PC, Salmon J, Gilkeson G, Holers VM. Thurman JM, et al. Mol Immunol. 2005 Jan;42(1):87-97. doi: 10.1016/j.molimm.2004.07.043. Mol Immunol. 2005. PMID: 15488947

8

Structure-based mapping of DAF active site residues that accelerate the decay of C3 convertases.

Kuttner-Kondo L, Hourcade DE, Anderson VE, Muqim N, Mitchell L, Soares DC, Barlow PN, Medof ME. Kuttner-Kondo L, et al. Among authors: mitchell l. J Biol Chem. 2007 Jun 22;282(25):18552-18562. doi: 10.1074/jbc.M611650200. Epub 2007 Mar 29. J Biol Chem. 2007. PMID: 17395591 Free article.

9

Properdin can initiate complement activation by binding specific target surfaces and providing a platform for de novo convertase assembly.

Spitzer D, Mitchell LM, Atkinson JP, Hourcade DE. Spitzer D, et al. J Immunol. 2007 Aug 15;179(4):2600-8. doi: 10.4049/jimmunol.179.4.2600. J Immunol. 2007. PMID: 17675523

10

The complement protein properdin binds apoptotic T cells and promotes complement activation and phagocytosis.

Kemper C, Mitchell LM, Zhang L, Hourcade DE. Kemper C, et al. Among authors: mitchell lm. Proc Natl Acad Sci U S A. 2008 Jul 1;105(26):9023-8. doi: 10.1073/pnas.0801015105. Epub 2008 Jun 25. Proc Natl Acad Sci U S A. 2008. PMID: 18579773 Free PMC article.

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