Sato K - Search Results

1

Altered oligomeric states in pathogenic ALS2 variants associated with juvenile motor neuron diseases cause loss of ALS2-mediated endosomal function.

Sato K, Otomo A, Ueda MT, Hiratsuka Y, Suzuki-Utsunomiya K, Sugiyama J, Murakoshi S, Mitsui S, Ono S, Nakagawa S, Shang HF, Hadano S. Sato K, et al. J Biol Chem. 2018 Nov 2;293(44):17135-17153. doi: 10.1074/jbc.RA118.003849. Epub 2018 Sep 17. J Biol Chem. 2018. PMID: 30224357 Free PMC article.

2

Functional links between SQSTM1 and ALS2 in the pathogenesis of ALS: cumulative impact on the protection against mutant SOD1-mediated motor dysfunction in mice.

Hadano S, Mitsui S, Pan L, Otomo A, Kubo M, Sato K, Ono S, Onodera W, Abe K, Chen X, Koike M, Uchiyama Y, Aoki M, Warabi E, Yamamoto M, Ishii T, Yanagawa T, Shang HF, Yoshii F. Hadano S, et al. Among authors: sato k. Hum Mol Genet. 2016 Aug 1;25(15):3321-3340. doi: 10.1093/hmg/ddw180. Epub 2016 Jul 20. Hum Mol Genet. 2016. PMID: 27439389

3

Systemic overexpression of SQSTM1/p62 accelerates disease onset in a SOD1^H46R-expressing ALS mouse model.

Mitsui S, Otomo A, Nozaki M, Ono S, Sato K, Shirakawa R, Adachi H, Aoki M, Sobue G, Shang HF, Hadano S. Mitsui S, et al. Among authors: sato k. Mol Brain. 2018 May 29;11(1):30. doi: 10.1186/s13041-018-0373-8. Mol Brain. 2018. PMID: 29843805 Free PMC article.

4

ALS2, the small GTPase Rab17-interacting protein, regulates maturation and sorting of Rab17-associated endosomes.

Ono S, Otomo A, Murakoshi S, Mitsui S, Sato K, Fukuda M, Hadano S. Ono S, et al. Among authors: sato k. Biochem Biophys Res Commun. 2020 Mar 19;523(4):908-915. doi: 10.1016/j.bbrc.2019.12.122. Epub 2020 Jan 17. Biochem Biophys Res Commun. 2020. PMID: 31959474

5

SQSTM1^L341V variant that is linked to sporadic ALS exhibits impaired association with MAP1LC3 in cultured cells.

Nozaki M, Otomo A, Mitsui S, Ono S, Shirakawa R, Chen Y, Hama Y, Sato K, Chen X, Suzuki T, Shang HF, Hadano S. Nozaki M, et al. Among authors: sato k. eNeurologicalSci. 2020 Nov 30;22:100301. doi: 10.1016/j.ensci.2020.100301. eCollection 2021 Mar. eNeurologicalSci. 2020. PMID: 33319079 Free PMC article.

6

The N-terminal intrinsically disordered region mediates intracellular localization and self-oligomerization of ALS2.

Shimakura K, Sato K, Mitsui S, Ono S, Otomo A, Hadano S. Shimakura K, et al. Among authors: sato k. Biochem Biophys Res Commun. 2021 Sep 10;569:106-111. doi: 10.1016/j.bbrc.2021.06.095. Epub 2021 Jul 6. Biochem Biophys Res Commun. 2021. PMID: 34243065

7

High-throughput quantitative analysis of axonal transport in cultured neurons from SOD1^H46R ALS mice by using a microfluidic device.

Otomo A, Ono S, Sato K, Mitsui S, Shimakura K, Kimura H, Hadano S. Otomo A, et al. Among authors: sato k. Neurosci Res. 2022 Jan;174:46-52. doi: 10.1016/j.neures.2021.07.005. Epub 2021 Aug 2. Neurosci Res. 2022. PMID: 34352295 Free article.

8

SQSTM1, a protective factor of SOD1-linked motor neuron disease, regulates the accumulation and distribution of ubiquitinated protein aggregates in neuron.

Mitsui S, Otomo A, Sato K, Ishiyama M, Shimakura K, Okada-Yamaguchi C, Warabi E, Yanagawa T, Aoki M, Shang HF, Hadano S. Mitsui S, et al. Among authors: sato k. Neurochem Int. 2022 Sep;158:105364. doi: 10.1016/j.neuint.2022.105364. Epub 2022 May 28. Neurochem Int. 2022. PMID: 35640762 Free article.

9

Personalized Treatment for Infantile Ascending Hereditary Spastic Paralysis Based on In Silico Strategies.

Rossi Sebastiano M, Ermondi G, Sato K, Otomo A, Hadano S, Caron G. Rossi Sebastiano M, et al. Among authors: sato k. Molecules. 2022 Oct 19;27(20):7063. doi: 10.3390/molecules27207063. Molecules. 2022. PMID: 36296656 Free PMC article.

10

Central nervous system specific high molecular weight ALS2/alsin homophilic complex is enriched in mouse brain synaptosomes.

Sato K, Suzuki-Utsunomiya K, Mitsui S, Ono S, Shimakura K, Otomo A, Hadano S. Sato K, et al. Biochem Biophys Res Commun. 2023 Jan 1;638:168-175. doi: 10.1016/j.bbrc.2022.11.061. Epub 2022 Nov 22. Biochem Biophys Res Commun. 2023. PMID: 36459881 Free article.

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