Callender JA - Search Results

1

Protein kinase Cα gain-of-function variant in Alzheimer's disease displays enhanced catalysis by a mechanism that evades down-regulation.

Callender JA, Yang Y, Lordén G, Stephenson NL, Jones AC, Brognard J, Newton AC. Callender JA, et al. Proc Natl Acad Sci U S A. 2018 Jun 12;115(24):E5497-E5505. doi: 10.1073/pnas.1805046115. Epub 2018 May 29. Proc Natl Acad Sci U S A. 2018. PMID: 29844158 Free PMC article.

2

Intramolecular C2 Domain-Mediated Autoinhibition of Protein Kinase C βII.

Antal CE, Callender JA, Kornev AP, Taylor SS, Newton AC. Antal CE, et al. Among authors: callender ja. Cell Rep. 2015 Aug 25;12(8):1252-60. doi: 10.1016/j.celrep.2015.07.039. Epub 2015 Aug 13. Cell Rep. 2015. PMID: 26279568 Free PMC article.

3

Protein kinase C beta II suppresses colorectal cancer by regulating IGF-1 mediated cell survival.

Dowling CM, Phelan J, Callender JA, Cathcart MC, Mehigan B, McCormick P, Dalton T, Coffey JC, Newton AC, O'Sullivan J, Kiely PA. Dowling CM, et al. Among authors: callender ja. Oncotarget. 2016 Apr 12;7(15):20919-33. doi: 10.18632/oncotarget.8062. Oncotarget. 2016. PMID: 26989024 Free PMC article.

4

Gain-of-function mutations in protein kinase Cα (PKCα) may promote synaptic defects in Alzheimer's disease.

Alfonso SI, Callender JA, Hooli B, Antal CE, Mullin K, Sherman MA, Lesné SE, Leitges M, Newton AC, Tanzi RE, Malinow R. Alfonso SI, et al. Among authors: callender ja. Sci Signal. 2016 May 10;9(427):ra47. doi: 10.1126/scisignal.aaf6209. Sci Signal. 2016. PMID: 27165780 Free PMC article.

5

Conventional protein kinase C in the brain: 40 years later.

Callender JA, Newton AC. Callender JA, et al. Neuronal Signal. 2017 Apr 10;1(2):NS20160005. doi: 10.1042/NS20160005. eCollection 2017 Apr. Neuronal Signal. 2017. PMID: 32714576 Free PMC article. Review.

6

Defective Transcytosis of APP and Lipoproteins in Human iPSC-Derived Neurons with Familial Alzheimer's Disease Mutations.

Woodruff G, Reyna SM, Dunlap M, Van Der Kant R, Callender JA, Young JE, Roberts EA, Goldstein LS. Woodruff G, et al. Among authors: callender ja. Cell Rep. 2016 Oct 11;17(3):759-773. doi: 10.1016/j.celrep.2016.09.034. Cell Rep. 2016. PMID: 27732852 Free PMC article.

7

Prion protein post-translational modifications modulate heparan sulfate binding and limit aggregate size in prion disease.

Callender JA, Sevillano AM, Soldau K, Kurt TD, Schumann T, Pizzo DP, Altmeppen H, Glatzel M, Esko JD, Sigurdson CJ. Callender JA, et al. Neurobiol Dis. 2020 Aug;142:104955. doi: 10.1016/j.nbd.2020.104955. Epub 2020 May 24. Neurobiol Dis. 2020. PMID: 32454127 Free PMC article.

8

Short and sweet: How glycans impact prion conversion, cofactor interactions, and cross-species transmission.

Aguilar-Calvo P, Callender JA, Sigurdson CJ. Aguilar-Calvo P, et al. Among authors: callender ja. PLoS Pathog. 2021 Jan 14;17(1):e1009123. doi: 10.1371/journal.ppat.1009123. eCollection 2021 Jan. PLoS Pathog. 2021. PMID: 33444414 Free PMC article. No abstract available.

9

Diminished Neuronal ESCRT-0 Function Exacerbates AMPA Receptor Derangement and Accelerates Prion-Induced Neurodegeneration.

Lawrence JA, Aguilar-Calvo P, Ojeda-Juárez D, Khuu H, Soldau K, Pizzo DP, Wang J, Malik A, Shay TF, Sullivan EE, Aulston B, Song SM, Callender JA, Sanchez H, Geschwind MD, Roy S, Rissman RA, Trejo J, Tanaka N, Wu C, Chen X, Patrick GN, Sigurdson CJ. Lawrence JA, et al. Among authors: callender ja. J Neurosci. 2023 May 24;43(21):3970-3984. doi: 10.1523/JNEUROSCI.1878-22.2023. Epub 2023 Apr 5. J Neurosci. 2023. PMID: 37019623 Free PMC article.

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