Petit PX - Search Results

1

Decreasing cytosolic translation is beneficial to yeast and human Tafazzin-deficient cells.

de Taffin de Tilques M, Lasserre JP, Godard F, Sardin E, Bouhier M, Le Guedard M, Kucharczyk R, Petit PX, Testet E, di Rago JP, Tribouillard-Tanvier D. de Taffin de Tilques M, et al. Among authors: petit px. Microb Cell. 2018 Feb 18;5(5):220-232. doi: 10.15698/mic2018.05.629. Microb Cell. 2018. PMID: 29796387 Free PMC article.

2

Barth syndrome: cellular compensation of mitochondrial dysfunction and apoptosis inhibition due to changes in cardiolipin remodeling linked to tafazzin (TAZ) gene mutation.

Gonzalvez F, D'Aurelio M, Boutant M, Moustapha A, Puech JP, Landes T, Arnauné-Pelloquin L, Vial G, Taleux N, Slomianny C, Wanders RJ, Houtkooper RH, Bellenguer P, Møller IM, Gottlieb E, Vaz FM, Manfredi G, Petit PX. Gonzalvez F, et al. Among authors: petit px. Biochim Biophys Acta. 2013 Aug;1832(8):1194-206. doi: 10.1016/j.bbadis.2013.03.005. Epub 2013 Mar 20. Biochim Biophys Acta. 2013. PMID: 23523468 Free article.

3

Tafazzin Mutation Affecting Cardiolipin Leads to Increased Mitochondrial Superoxide Anions and Mitophagy Inhibition in Barth Syndrome.

Petit PX, Ardilla-Osorio H, Penalvia L, Rainey NE. Petit PX, et al. Cells. 2020 Oct 21;9(10):2333. doi: 10.3390/cells9102333. Cells. 2020. PMID: 33096711 Free PMC article.

4

Barth Syndrome: From Mitochondrial Dysfunctions Associated with Aberrant Production of Reactive Oxygen Species to Pluripotent Stem Cell Studies.

Saric A, Andreau K, Armand AS, Møller IM, Petit PX. Saric A, et al. Among authors: petit px. Front Genet. 2016 Jan 20;6:359. doi: 10.3389/fgene.2015.00359. eCollection 2015. Front Genet. 2016. PMID: 26834781 Free PMC article. Review.

5

Role of cardiolipin on tBid and tBid/Bax synergistic effects on yeast mitochondria.

Gonzalvez F, Bessoule JJ, Rocchiccioli F, Manon S, Petit PX. Gonzalvez F, et al. Among authors: petit px. Cell Death Differ. 2005 Jun;12(6):659-67. doi: 10.1038/sj.cdd.4401585. Cell Death Differ. 2005. PMID: 15818414

6

Giant unilamellar vesicles (GUVs) as a new tool for analysis of caspase-8/Bid-FL complex binding to cardiolipin and its functional activity.

Jalmar O, García-Sáez AJ, Berland L, Gonzalvez F, Petit PX. Jalmar O, et al. Among authors: petit px. Cell Death Dis. 2010 Dec 2;1(12):e103. doi: 10.1038/cddis.2010.81. Cell Death Dis. 2010. PMID: 21368876 Free PMC article.

7

Cardiolipin provides an essential activating platform for caspase-8 on mitochondria.

Gonzalvez F, Schug ZT, Houtkooper RH, MacKenzie ED, Brooks DG, Wanders RJ, Petit PX, Vaz FM, Gottlieb E. Gonzalvez F, et al. Among authors: petit px. J Cell Biol. 2008 Nov 17;183(4):681-96. doi: 10.1083/jcb.200803129. Epub 2008 Nov 10. J Cell Biol. 2008. PMID: 19001123 Free PMC article.

8

Interaction of the alpha-helical H6 peptide from the pro-apoptotic protein tBid with cardiolipin.

Petit PX, Dupaigne P, Pariselli F, Gonzalvez F, Etienne F, Rameau C, Bernard S. Petit PX, et al. FEBS J. 2009 Nov;276(21):6338-54. doi: 10.1111/j.1742-4658.2009.07345.x. Epub 2009 Oct 1. FEBS J. 2009. PMID: 19796174 Free article.

9

Curcumin, a Multifaceted Hormetic Agent, Mediates an Intricate Crosstalk between Mitochondrial Turnover, Autophagy, and Apoptosis.

Rainey NE, Moustapha A, Petit PX. Rainey NE, et al. Among authors: petit px. Oxid Med Cell Longev. 2020 Jul 18;2020:3656419. doi: 10.1155/2020/3656419. eCollection 2020. Oxid Med Cell Longev. 2020. PMID: 32765806 Free PMC article. Review.

10

Synergistic cellular effects including mitochondrial destabilization, autophagy and apoptosis following low-level exposure to a mixture of lipophilic persistent organic pollutants.

Rainey NE, Saric A, Leberre A, Dewailly E, Slomianny C, Vial G, Zeliger HI, Petit PX. Rainey NE, et al. Among authors: petit px. Sci Rep. 2017 Jul 5;7(1):4728. doi: 10.1038/s41598-017-04654-0. Sci Rep. 2017. PMID: 28680151 Free PMC article.

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