Ott T - Search Results

1

Characterization of Targeted Connexin32-Deficient Mice: A Model for the Human Charcot-Marie-Tooth (X-Type) Inherited Disease.

Willecke K, Temme A, Teubner B, Ott T. Willecke K, et al. Among authors: ott t. Ann N Y Acad Sci. 1999 Oct;883(1):302-309. doi: 10.1111/j.1749-6632.1999.tb08592.x. Ann N Y Acad Sci. 1999. PMID: 29086934 No abstract available.

2

Characterization of targeted connexin32-deficient mice: a model for the human Charcot-Marie-Tooth (X-type) inherited disease.

Willecke K, Temme A, Teubner B, Ott T. Willecke K, et al. Among authors: ott t. Ann N Y Acad Sci. 1999 Sep 14;883:302-9. Ann N Y Acad Sci. 1999. PMID: 10586255 Review. No abstract available.

3

Morphology and morphometric investigation of hepatocellular preneoplastic lesions and neoplasms in connexin32-deficient mice.

Evert M, Ott T, Temme A, Willecke K, Dombrowski F. Evert M, et al. Among authors: ott t. Carcinogenesis. 2002 May;23(5):697-703. doi: 10.1093/carcin/23.5.697. Carcinogenesis. 2002. PMID: 12016140

4

Altered connexin expression and wound healing in the epidermis of connexin-deficient mice.

Kretz M, Euwens C, Hombach S, Eckardt D, Teubner B, Traub O, Willecke K, Ott T. Kretz M, et al. Among authors: ott t. J Cell Sci. 2003 Aug 15;116(Pt 16):3443-52. doi: 10.1242/jcs.00638. Epub 2003 Jul 2. J Cell Sci. 2003. PMID: 12840073

5

The extent of synchronous initiation and termination of DNA synthesis in regenerating mouse liver is dependent on connexin32 expressing gap junctions.

Temme A, Ott T, Dombrowski F, Willecke K. Temme A, et al. Among authors: ott t. J Hepatol. 2000 Apr;32(4):627-35. doi: 10.1016/s0168-8278(00)80225-1. J Hepatol. 2000. PMID: 10782912

6

Acute-phase response and circadian expression of connexin26 are not altered in connexin32-deficient mouse liver.

Temme A, Ott T, Haberberger T, Traub O, Willecke K. Temme A, et al. Among authors: ott t. Cell Tissue Res. 2000 Apr;300(1):111-7. doi: 10.1007/s004410000177. Cell Tissue Res. 2000. PMID: 10805080

7

Biological functions of connexin genes revealed by human genetic defects, dominant negative approaches and targeted deletions in the mouse.

Willecke K, Kirchhoff S, Plum A, Temme A, Thönnissen E, Ott T. Willecke K, et al. Among authors: ott t. Novartis Found Symp. 1999;219:76-88; discussion 88-96. doi: 10.1002/9780470515587.ch6. Novartis Found Symp. 1999. PMID: 10207899 Review.

8

The effect of connexin32 null mutation on hepatocarcinogenesis in different mouse strains.

Moennikes O, Buchmann A, Ott T, Willecke1 K, Schwarz M. Moennikes O, et al. Among authors: ott t. Carcinogenesis. 1999 Jul;20(7):1379-82. doi: 10.1093/carcin/20.7.1379. Carcinogenesis. 1999. PMID: 10383916

9

Quantitative analysis of gap-junctional intercellular communication in precision-cut mouse liver slices.

Romualdi A, Niessen H, Dombrowski F, Willecke K, Ott T. Romualdi A, et al. Among authors: ott t. Cell Tissue Res. 2002 Mar;307(3):315-20. doi: 10.1007/s00441-002-0518-y. Epub 2002 Feb 27. Cell Tissue Res. 2002. PMID: 11904767

10

Dilated bile canaliculi and attenuated decrease of nerve-dependent bile secretion in connexin32-deficient mouse liver.

Temme A, Stümpel F, Söhl G, Rieber EP, Jungermann K, Willecke K, Ott T. Temme A, et al. Among authors: ott t. Pflugers Arch. 2001 Sep;442(6):961-6. doi: 10.1007/s004240100623. Pflugers Arch. 2001. PMID: 11680630

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