Li L - Search Results

1

The persistence of low-grade inflammatory monocytes contributes to aggravated atherosclerosis.

Geng S, Chen K, Yuan R, Peng L, Maitra U, Diao N, Chen C, Zhang Y, Hu Y, Qi CF, Pierce S, Ling W, Xiong H, Li L. Geng S, et al. Among authors: li l. Nat Commun. 2016 Nov 8;7:13436. doi: 10.1038/ncomms13436. Nat Commun. 2016. PMID: 27824038 Free PMC article.

2

Differential regulation of Foxp3 and IL-17 expression in CD4 T helper cells by IRAK-1.

Maitra U, Davis S, Reilly CM, Li L. Maitra U, et al. Among authors: li l. J Immunol. 2009 May 1;182(9):5763-9. doi: 10.4049/jimmunol.0900124. J Immunol. 2009. PMID: 19380824 Free PMC article.

3

An innate immunity signaling process suppresses macrophage ABCA1 expression through IRAK-1-mediated downregulation of retinoic acid receptor alpha and NFATc2.

Maitra U, Parks JS, Li L. Maitra U, et al. Among authors: li l. Mol Cell Biol. 2009 Nov;29(22):5989-97. doi: 10.1128/MCB.00541-09. Epub 2009 Sep 14. Mol Cell Biol. 2009. PMID: 19752193 Free PMC article.

4

Molecular mechanism underlying the suppression of lipid oxidation during endotoxemia.

Maitra U, Chang S, Singh N, Li L. Maitra U, et al. Among authors: li l. Mol Immunol. 2009 Dec;47(2-3):420-5. doi: 10.1016/j.molimm.2009.08.023. Epub 2009 Sep 20. Mol Immunol. 2009. PMID: 19773084 Free PMC article.

5

IRAK-1 contributes to lipopolysaccharide-induced reactive oxygen species generation in macrophages by inducing NOX-1 transcription and Rac1 activation and suppressing the expression of antioxidative enzymes.

Maitra U, Singh N, Gan L, Ringwood L, Li L. Maitra U, et al. Among authors: li l. J Biol Chem. 2009 Dec 18;284(51):35403-11. doi: 10.1074/jbc.M109.059501. J Biol Chem. 2009. PMID: 19850916 Free PMC article.

6

Molecular mechanism underlying the inflammatory complication of leptin in macrophages.

Vaughan T, Li L. Vaughan T, et al. Among authors: li l. Mol Immunol. 2010 Sep;47(15):2515-8. doi: 10.1016/j.molimm.2010.06.006. Epub 2010 Jul 8. Mol Immunol. 2010. PMID: 20619458

7

Low-dose endotoxin induces inflammation by selectively removing nuclear receptors and activating CCAAT/enhancer-binding protein δ.

Maitra U, Gan L, Chang S, Li L. Maitra U, et al. Among authors: li l. J Immunol. 2011 Apr 1;186(7):4467-73. doi: 10.4049/jimmunol.1003300. Epub 2011 Feb 25. J Immunol. 2011. PMID: 21357541

8

Molecular mechanisms responsible for the selective and low-grade induction of proinflammatory mediators in murine macrophages by lipopolysaccharide.

Maitra U, Deng H, Glaros T, Baker B, Capelluto DG, Li Z, Li L. Maitra U, et al. Among authors: li z, li l. J Immunol. 2012 Jul 15;189(2):1014-23. doi: 10.4049/jimmunol.1200857. Epub 2012 Jun 15. J Immunol. 2012. PMID: 22706082 Free PMC article.

9

Molecular mechanisms responsible for the reduced expression of cholesterol transporters from macrophages by low-dose endotoxin.

Maitra U, Li L. Maitra U, et al. Among authors: li l. Arterioscler Thromb Vasc Biol. 2013 Jan;33(1):24-33. doi: 10.1161/ATVBAHA.112.300049. Epub 2012 Nov 1. Arterioscler Thromb Vasc Biol. 2013. PMID: 23117655 Free PMC article.

10

Molecular mechanism responsible for the priming of macrophage activation.

Deng H, Maitra U, Morris M, Li L. Deng H, et al. Among authors: li l. J Biol Chem. 2013 Feb 8;288(6):3897-906. doi: 10.1074/jbc.M112.424390. Epub 2012 Dec 21. J Biol Chem. 2013. PMID: 23264622 Free PMC article.

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