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"Good enough solutions" and the genetics of complex diseases.
Weiss JN, Karma A, MacLellan WR, Deng M, Rau CD, Rees CM, Wang J, Wisniewski N, Eskin E, Horvath S, Qu Z, Wang Y, Lusis AJ. Weiss JN, et al. Circ Res. 2012 Aug 3;111(4):493-504. doi: 10.1161/CIRCRESAHA.112.269084. Circ Res. 2012. PMID: 22859671 Free PMC article. Review.
Deletion of MLIP (muscle-enriched A-type lamin-interacting protein) leads to cardiac hyperactivation of Akt/mammalian target of rapamycin (mTOR) and impaired cardiac adaptation.
Cattin ME, Wang J, Weldrick JJ, Roeske CL, Mak E, Thorn SL, DaSilva JN, Wang Y, Lusis AJ, Burgon PG. Cattin ME, et al. Among authors: wang j, wang y. J Biol Chem. 2015 Oct 30;290(44):26699-714. doi: 10.1074/jbc.M115.678433. Epub 2015 Sep 10. J Biol Chem. 2015. PMID: 26359501 Free PMC article.
Relationship of disease-associated gene expression to cardiac phenotype is buffered by genetic diversity and chromatin regulation.
Karbassi E, Monte E, Chapski DJ, Lopez R, Rosa Garrido M, Kim J, Wisniewski N, Rau CD, Wang JJ, Weiss JN, Wang Y, Lusis AJ, Vondriska TM. Karbassi E, et al. Among authors: wang jj, wang y. Physiol Genomics. 2016 Aug 1;48(8):601-15. doi: 10.1152/physiolgenomics.00035.2016. Epub 2016 Jun 10. Physiol Genomics. 2016. PMID: 27287924 Free PMC article.
5,290 results