McKinsey TA - Search Results

1

Discovery of novel small molecule inhibitors of cardiac hypertrophy using high throughput, high content imaging.

Reid BG, Stratton MS, Bowers S, Cavasin MA, Demos-Davies KM, Susano I, McKinsey TA. Reid BG, et al. Among authors: mckinsey ta. J Mol Cell Cardiol. 2016 Aug;97:106-13. doi: 10.1016/j.yjmcc.2016.04.015. Epub 2016 Apr 27. J Mol Cell Cardiol. 2016. PMID: 27130278 Free PMC article.

2

The CRM1 nuclear export receptor controls pathological cardiac gene expression.

Harrison BC, Roberts CR, Hood DB, Sweeney M, Gould JM, Bush EW, McKinsey TA. Harrison BC, et al. Among authors: mckinsey ta. Mol Cell Biol. 2004 Dec;24(24):10636-49. doi: 10.1128/MCB.24.24.10636-10649.2004. Mol Cell Biol. 2004. PMID: 15572669 Free PMC article.

3

Small-molecule therapies for cardiac hypertrophy: moving beneath the cell surface.

McKinsey TA, Kass DA. McKinsey TA, et al. Nat Rev Drug Discov. 2007 Aug;6(8):617-35. doi: 10.1038/nrd2193. Nat Rev Drug Discov. 2007. PMID: 17643091 Review.

4

Suppression of HDAC nuclear export and cardiomyocyte hypertrophy by novel irreversible inhibitors of CRM1.

Monovich L, Koch KA, Burgis R, Osimboni E, Mann T, Wall D, Gao J, Feng Y, Vega RB, Turner BA, Hood DB, Law A, Papst PJ, Koditek D, Chapo JA, Reid BG, Melvin LS, Pagratis NC, McKinsey TA. Monovich L, et al. Among authors: mckinsey ta. Biochim Biophys Acta. 2009 May;1789(5):422-31. doi: 10.1016/j.bbagrm.2009.04.001. Epub 2009 May 3. Biochim Biophys Acta. 2009. PMID: 19414071

5

Isoform-selective HDAC inhibitors: closing in on translational medicine for the heart.

McKinsey TA. McKinsey TA. J Mol Cell Cardiol. 2011 Oct;51(4):491-6. doi: 10.1016/j.yjmcc.2010.11.009. Epub 2010 Nov 23. J Mol Cell Cardiol. 2011. PMID: 21108947 Review.

6

Cardiac HDAC6 catalytic activity is induced in response to chronic hypertension.

Lemon DD, Horn TR, Cavasin MA, Jeong MY, Haubold KW, Long CS, Irwin DC, McCune SA, Chung E, Leinwand LA, McKinsey TA. Lemon DD, et al. Among authors: mckinsey ta. J Mol Cell Cardiol. 2011 Jul;51(1):41-50. doi: 10.1016/j.yjmcc.2011.04.005. Epub 2011 Apr 23. J Mol Cell Cardiol. 2011. PMID: 21539845 Free PMC article.

7

A novel protein kinase C target site in protein kinase D is phosphorylated in response to signals for cardiac hypertrophy.

Phan D, Stratton MS, Huynh QK, McKinsey TA. Phan D, et al. Among authors: mckinsey ta. Biochem Biophys Res Commun. 2011 Jul 29;411(2):335-41. doi: 10.1016/j.bbrc.2011.06.143. Epub 2011 Jun 25. Biochem Biophys Res Commun. 2011. PMID: 21726532

8

Selective class I histone deacetylase inhibition suppresses hypoxia-induced cardiopulmonary remodeling through an antiproliferative mechanism.

Cavasin MA, Demos-Davies K, Horn TR, Walker LA, Lemon DD, Birdsey N, Weiser-Evans MC, Harral J, Irwin DC, Anwar A, Yeager ME, Li M, Watson PA, Nemenoff RA, Buttrick PM, Stenmark KR, McKinsey TA. Cavasin MA, et al. Among authors: mckinsey ta. Circ Res. 2012 Mar 2;110(5):739-48. doi: 10.1161/CIRCRESAHA.111.258426. Epub 2012 Jan 26. Circ Res. 2012. PMID: 22282194 Free PMC article.

9

Roles of histone deacetylation and AMP kinase in regulation of cardiomyocyte PGC-1α gene expression in hypoxia.

Ramjiawan A, Bagchi RA, Blant A, Albak L, Cavasin MA, Horn TR, McKinsey TA, Czubryt MP. Ramjiawan A, et al. Among authors: mckinsey ta. Am J Physiol Cell Physiol. 2013 Jun 1;304(11):C1064-72. doi: 10.1152/ajpcell.00262.2012. Epub 2013 Mar 20. Am J Physiol Cell Physiol. 2013. PMID: 23515531 Free article.

10

Signal-dependent repression of DUSP5 by class I HDACs controls nuclear ERK activity and cardiomyocyte hypertrophy.

Ferguson BS, Harrison BC, Jeong MY, Reid BG, Wempe MF, Wagner FF, Holson EB, McKinsey TA. Ferguson BS, et al. Among authors: mckinsey ta. Proc Natl Acad Sci U S A. 2013 Jun 11;110(24):9806-11. doi: 10.1073/pnas.1301509110. Epub 2013 May 29. Proc Natl Acad Sci U S A. 2013. PMID: 23720316 Free PMC article.

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