Xing B - Search Results

1

MW151 Inhibited IL-1β Levels after Traumatic Brain Injury with No Effect on Microglia Physiological Responses.

Bachstetter AD, Zhou Z, Rowe RK, Xing B, Goulding DS, Conley AN, Sompol P, Meier S, Abisambra JF, Lifshitz J, Watterson DM, Van Eldik LJ. Bachstetter AD, et al. Among authors: xing b. PLoS One. 2016 Feb 12;11(2):e0149451. doi: 10.1371/journal.pone.0149451. eCollection 2016. PLoS One. 2016. PMID: 26871438 Free PMC article.

2

Microglial p38α MAPK is a key regulator of proinflammatory cytokine up-regulation induced by toll-like receptor (TLR) ligands or beta-amyloid (Aβ).

Bachstetter AD, Xing B, de Almeida L, Dimayuga ER, Watterson DM, Van Eldik LJ. Bachstetter AD, et al. Among authors: xing b. J Neuroinflammation. 2011 Jul 6;8:79. doi: 10.1186/1742-2094-8-79. J Neuroinflammation. 2011. PMID: 21733175 Free PMC article.

3

Deficiency in p38β MAPK fails to inhibit cytokine production or protect neurons against inflammatory insult in in vitro and in vivo mouse models.

Xing B, Bachstetter AD, Van Eldik LJ. Xing B, et al. PLoS One. 2013;8(2):e56852. doi: 10.1371/journal.pone.0056852. Epub 2013 Feb 15. PLoS One. 2013. PMID: 23457629 Free PMC article.

4

Microglial p38α MAPK is critical for LPS-induced neuron degeneration, through a mechanism involving TNFα.

Xing B, Bachstetter AD, Van Eldik LJ. Xing B, et al. Mol Neurodegener. 2011 Dec 20;6:84. doi: 10.1186/1750-1326-6-84. Mol Neurodegener. 2011. PMID: 22185458 Free PMC article.

5

Development of Novel In Vivo Chemical Probes to Address CNS Protein Kinase Involvement in Synaptic Dysfunction.

Watterson DM, Grum-Tokars VL, Roy SM, Schavocky JP, Bradaric BD, Bachstetter AD, Xing B, Dimayuga E, Saeed F, Zhang H, Staniszewski A, Pelletier JC, Minasov G, Anderson WF, Arancio O, Van Eldik LJ. Watterson DM, et al. Among authors: xing b. PLoS One. 2013 Jun 26;8(6):e66226. doi: 10.1371/journal.pone.0066226. Print 2013. PLoS One. 2013. PMID: 23840427 Free PMC article.

6

Inhibition of neuronal p38α, but not p38β MAPK, provides neuroprotection against three different neurotoxic insults.

Xing B, Bachstetter AD, Van Eldik LJ. Xing B, et al. J Mol Neurosci. 2015 Feb;55(2):509-18. doi: 10.1007/s12031-014-0372-x. Epub 2014 Jul 11. J Mol Neurosci. 2015. PMID: 25012593 Free PMC article.

7

The p38alpha mitogen-activated protein kinase limits the CNS proinflammatory cytokine response to systemic lipopolysaccharide, potentially through an IL-10 dependent mechanism.

Bachstetter AD, Xing B, Van Eldik LJ. Bachstetter AD, et al. Among authors: xing b. J Neuroinflammation. 2014 Oct 10;11:175. doi: 10.1186/s12974-014-0175-6. J Neuroinflammation. 2014. PMID: 25297465 Free PMC article.

8

Glial cell line-derived neurotrophic factor protects midbrain dopaminergic neurons against lipopolysaccharide neurotoxicity.

Xing B, Xin T, Zhao L, Hunter RL, Chen Y, Bing G. Xing B, et al. J Neuroimmunol. 2010 Aug 25;225(1-2):43-51. doi: 10.1016/j.jneuroim.2010.04.010. Epub 2010 May 14. J Neuroimmunol. 2010. PMID: 20471698 Free PMC article.

9

Pioglitazone inhibition of lipopolysaccharide-induced nitric oxide synthase is associated with altered activity of p38 MAP kinase and PI3K/Akt.

Xing B, Xin T, Hunter RL, Bing G. Xing B, et al. J Neuroinflammation. 2008 Jan 18;5:4. doi: 10.1186/1742-2094-5-4. J Neuroinflammation. 2008. PMID: 18205920 Free PMC article.

10

Neuroprotection with pioglitazone against LPS insult on dopaminergic neurons may be associated with its inhibition of NF-kappaB and JNK activation and suppression of COX-2 activity.

Xing B, Liu M, Bing G. Xing B, et al. J Neuroimmunol. 2007 Dec;192(1-2):89-98. doi: 10.1016/j.jneuroim.2007.09.029. Epub 2007 Oct 31. J Neuroimmunol. 2007. PMID: 17976742

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