Son M - Search Results

1

Copper delivery to the CNS by CuATSM effectively treats motor neuron disease in SOD(G93A) mice co-expressing the Copper-Chaperone-for-SOD.

Williams JR, Trias E, Beilby PR, Lopez NI, Labut EM, Bradford CS, Roberts BR, McAllum EJ, Crouch PJ, Rhoads TW, Pereira C, Son M, Elliott JL, Franco MC, Estévez AG, Barbeito L, Beckman JS. Williams JR, et al. Among authors: son m. Neurobiol Dis. 2016 May;89:1-9. doi: 10.1016/j.nbd.2016.01.020. Epub 2016 Jan 27. Neurobiol Dis. 2016. PMID: 26826269 Free PMC article.

2

Redox susceptibility of SOD1 mutants is associated with the differential response to CCS over-expression in vivo.

Son M, Fu Q, Puttaparthi K, Matthews CM, Elliott JL. Son M, et al. Neurobiol Dis. 2009 Apr;34(1):155-62. doi: 10.1016/j.nbd.2009.01.005. Neurobiol Dis. 2009. PMID: 19320055 Free PMC article.

3

Mitochondrial defects in transgenic mice expressing Cu,Zn superoxide dismutase mutations: the role of copper chaperone for SOD1.

Son M, Elliott JL. Son M, et al. J Neurol Sci. 2014 Jan 15;336(1-2):1-7. doi: 10.1016/j.jns.2013.11.004. Epub 2013 Nov 13. J Neurol Sci. 2014. PMID: 24269091 Review.

4

Overexpression of CCS in G93A-SOD1 mice leads to accelerated neurological deficits with severe mitochondrial pathology.

Son M, Puttaparthi K, Kawamata H, Rajendran B, Boyer PJ, Manfredi G, Elliott JL. Son M, et al. Proc Natl Acad Sci U S A. 2007 Apr 3;104(14):6072-7. doi: 10.1073/pnas.0610923104. Epub 2007 Mar 26. Proc Natl Acad Sci U S A. 2007. PMID: 17389365 Free PMC article.

5

Isolated cytochrome c oxidase deficiency in G93A SOD1 mice overexpressing CCS protein.

Son M, Leary SC, Romain N, Pierrel F, Winge DR, Haller RG, Elliott JL. Son M, et al. J Biol Chem. 2008 May 2;283(18):12267-75. doi: 10.1074/jbc.M708523200. Epub 2008 Mar 11. J Biol Chem. 2008. PMID: 18334481 Free PMC article.

6

Biochemical properties and in vivo effects of the SOD1 zinc-binding site mutant (H80G).

Son M, Srikanth U, Puttaparthi K, Luther C, Elliott JL. Son M, et al. J Neurochem. 2011 Sep;118(5):891-901. doi: 10.1111/j.1471-4159.2011.07360.x. Epub 2011 Jul 21. J Neurochem. 2011. PMID: 21692800 Free PMC article.

7

Disease progression in a transgenic model of familial amyotrophic lateral sclerosis is dependent on both neuronal and non-neuronal zinc binding proteins.

Puttaparthi K, Gitomer WL, Krishnan U, Son M, Rajendran B, Elliott JL. Puttaparthi K, et al. Among authors: son m. J Neurosci. 2002 Oct 15;22(20):8790-6. doi: 10.1523/JNEUROSCI.22-20-08790.2002. J Neurosci. 2002. PMID: 12388585 Free PMC article.

8

Novel mutations that enhance or repress the aggregation potential of SOD1.

Krishnan U, Son M, Rajendran B, Elliott JL. Krishnan U, et al. Among authors: son m. Mol Cell Biochem. 2006 Jul;287(1-2):201-11. doi: 10.1007/s11010-005-9112-4. Epub 2006 Apr 1. Mol Cell Biochem. 2006. PMID: 16583143

9

Aggregate formation in Cu,Zn superoxide dismutase-related proteins.

Son M, Cloyd CD, Rothstein JD, Rajendran B, Elliott JL. Son M, et al. J Biol Chem. 2003 Apr 18;278(16):14331-6. doi: 10.1074/jbc.M211698200. Epub 2003 Jan 27. J Biol Chem. 2003. PMID: 12551935 Free article.

10

Biological effects of CCS in the absence of SOD1 enzyme activation: implications for disease in a mouse model for ALS.

Proescher JB, Son M, Elliott JL, Culotta VC. Proescher JB, et al. Among authors: son m. Hum Mol Genet. 2008 Jun 15;17(12):1728-37. doi: 10.1093/hmg/ddn063. Epub 2008 Mar 12. Hum Mol Genet. 2008. PMID: 18337307 Free PMC article.

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