Samsa WE - Search Results

1

Deficiency of circadian clock protein BMAL1 in mice results in a low bone mass phenotype.

Samsa WE, Vasanji A, Midura RJ, Kondratov RV. Samsa WE, et al. Bone. 2016 Mar;84:194-203. doi: 10.1016/j.bone.2016.01.006. Epub 2016 Jan 14. Bone. 2016. PMID: 26789548 Free PMC article.

2

Circadian regulation of cell cycle: Molecular connections between aging and the circadian clock.

Khapre RV, Samsa WE, Kondratov RV. Khapre RV, et al. Among authors: samsa we. Ann Med. 2010 Sep;42(6):404-15. doi: 10.3109/07853890.2010.499134. Ann Med. 2010. PMID: 20568980 Free article. Review.

3

Deficiency of circadian protein CLOCK reduces lifespan and increases age-related cataract development in mice.

Dubrovsky YV, Samsa WE, Kondratov RV. Dubrovsky YV, et al. Among authors: samsa we. Aging (Albany NY). 2010 Dec;2(12):936-44. doi: 10.18632/aging.100241. Aging (Albany NY). 2010. PMID: 21149897 Free PMC article.

4

The master developmental regulator Jab1/Cops5/Csn5 is essential for proper bone growth and survival in mice.

Samsa WE, Mamidi MK, Hausman BS, Bashur LA, Greenfield EM, Zhou G. Samsa WE, et al. Bone. 2021 Feb;143:115733. doi: 10.1016/j.bone.2020.115733. Epub 2020 Nov 4. Bone. 2021. PMID: 33157284 Free PMC article.

5

Transcriptional control of chondrocyte specification and differentiation.

Liu CF, Samsa WE, Zhou G, Lefebvre V. Liu CF, et al. Among authors: samsa we. Semin Cell Dev Biol. 2017 Feb;62:34-49. doi: 10.1016/j.semcdb.2016.10.004. Epub 2016 Oct 19. Semin Cell Dev Biol. 2017. PMID: 27771362 Free PMC article. Review.

6

Signaling pathways regulating cartilage growth plate formation and activity.

Samsa WE, Zhou X, Zhou G. Samsa WE, et al. Semin Cell Dev Biol. 2017 Feb;62:3-15. doi: 10.1016/j.semcdb.2016.07.008. Epub 2016 Jul 11. Semin Cell Dev Biol. 2017. PMID: 27418125 Free PMC article. Review.

7

Targeted and sustained Sox9 expression in mouse hypertrophic chondrocytes causes severe and spontaneous osteoarthritis by perturbing cartilage homeostasis.

Liang B, Mamidi MK, Samsa WE, Chen Y, Lee B, Zheng Q, Zhou G. Liang B, et al. Among authors: samsa we. Am J Transl Res. 2020 Mar 15;12(3):1056-1069. eCollection 2020. Am J Transl Res. 2020. PMID: 32269734 Free PMC article.

8

The crucial p53-dependent oncogenic role of JAB1 in osteosarcoma in vivo.

Samsa WE, Mamidi MK, Bashur LA, Elliott R, Miron A, Chen Y, Lee B, Greenfield EM, Chan R, Danielpour D, Zhou G. Samsa WE, et al. Oncogene. 2020 Jun;39(23):4581-4591. doi: 10.1038/s41388-020-1320-6. Epub 2020 May 10. Oncogene. 2020. PMID: 32390003 Free PMC article.

9

The transcriptional cofactor Jab1/Cops5 is crucial for BMP-mediated mouse chondrocyte differentiation by repressing p53 activity.

Mamidi MK, Samsa WE, Bashur LA, Chen Y, Chan R, Lee B, Zhou G. Mamidi MK, et al. Among authors: samsa we. J Cell Physiol. 2021 Aug;236(8):5686-5697. doi: 10.1002/jcp.30254. Epub 2021 Jan 3. J Cell Physiol. 2021. PMID: 33393086 Free PMC article.

10

The transcription co-factor JAB1/COPS5, serves as a potential oncogenic hub of human chondrosarcoma cells in vitro.

Mamidi MK, Samsa WE, Danielpour D, Chan R, Zhou G. Mamidi MK, et al. Among authors: samsa we. Am J Cancer Res. 2021 Oct 15;11(10):5063-5075. eCollection 2021. Am J Cancer Res. 2021. PMID: 34765312 Free PMC article.

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