Faden AI - Search Results

1

Ablation of the transcription factors E2F1-2 limits neuroinflammation and associated neurological deficits after contusive spinal cord injury.

Wu J, Sabirzhanov B, Stoica BA, Lipinski MM, Zhao Z, Zhao S, Ward N, Yang D, Faden AI. Wu J, et al. Among authors: faden ai. Cell Cycle. 2015;14(23):3698-712. doi: 10.1080/15384101.2015.1104436. Cell Cycle. 2015. PMID: 26505089 Free PMC article.

2

Ceramide induces neuronal apoptosis through the caspase-9/caspase-3 pathway.

Movsesyan VA, Yakovlev AG, Dabaghyan EA, Stoica BA, Faden AI. Movsesyan VA, et al. Among authors: faden ai. Biochem Biophys Res Commun. 2002 Nov 29;299(2):201-7. doi: 10.1016/s0006-291x(02)02593-7. Biochem Biophys Res Commun. 2002. PMID: 12437970

3

Ceramide-induced neuronal apoptosis is associated with dephosphorylation of Akt, BAD, FKHR, GSK-3beta, and induction of the mitochondrial-dependent intrinsic caspase pathway.

Stoica BA, Movsesyan VA, Lea PM 4th, Faden AI. Stoica BA, et al. Among authors: faden ai. Mol Cell Neurosci. 2003 Mar;22(3):365-82. doi: 10.1016/s1044-7431(02)00028-3. Mol Cell Neurosci. 2003. PMID: 12691738

4

Gene expression profile changes are commonly modulated across models and species after traumatic brain injury.

Natale JE, Ahmed F, Cernak I, Stoica B, Faden AI. Natale JE, et al. Among authors: faden ai. J Neurotrauma. 2003 Oct;20(10):907-27. doi: 10.1089/089771503770195777. J Neurotrauma. 2003. PMID: 14588109

5

Modulation of stretch-induced enhancement of neuronal NMDA receptor current by mGluR1 depends upon presence of glia.

Lea PM 4th, Custer SJ, Stoica BA, Faden AI. Lea PM 4th, et al. Among authors: faden ai. J Neurotrauma. 2003 Nov;20(11):1233-49. doi: 10.1089/089771503770802907. J Neurotrauma. 2003. PMID: 14651810

6

BOK and NOXA are essential mediators of p53-dependent apoptosis.

Yakovlev AG, Di Giovanni S, Wang G, Liu W, Stoica B, Faden AI. Yakovlev AG, et al. Among authors: faden ai. J Biol Chem. 2004 Jul 2;279(27):28367-74. doi: 10.1074/jbc.M313526200. Epub 2004 Apr 21. J Biol Chem. 2004. PMID: 15102863 Free article.

7

MGLuR5 activation reduces beta-amyloid-induced cell death in primary neuronal cultures and attenuates translocation of cytochrome c and apoptosis-inducing factor.

Movsesyan VA, Stoica BA, Faden AI. Movsesyan VA, et al. Among authors: faden ai. J Neurochem. 2004 Jun;89(6):1528-36. doi: 10.1111/j.1471-4159.2004.02451.x. J Neurochem. 2004. PMID: 15189356 Free article.

8

Caspase inhibitor z-DEVD-fmk attenuates calpain and necrotic cell death in vitro and after traumatic brain injury.

Knoblach SM, Alroy DA, Nikolaeva M, Cernak I, Stoica BA, Faden AI. Knoblach SM, et al. Among authors: faden ai. J Cereb Blood Flow Metab. 2004 Oct;24(10):1119-32. doi: 10.1097/01.WCB.0000138664.17682.32. J Cereb Blood Flow Metab. 2004. PMID: 15529012

9

Ceramide induces neuronal apoptosis through mitogen-activated protein kinases and causes release of multiple mitochondrial proteins.

Stoica BA, Movsesyan VA, Knoblach SM, Faden AI. Stoica BA, et al. Among authors: faden ai. Mol Cell Neurosci. 2005 Jul;29(3):355-71. doi: 10.1016/j.mcn.2005.02.009. Mol Cell Neurosci. 2005. PMID: 15905098

10

Cell cycle inhibition provides neuroprotection and reduces glial proliferation and scar formation after traumatic brain injury.

Di Giovanni S, Movsesyan V, Ahmed F, Cernak I, Schinelli S, Stoica B, Faden AI. Di Giovanni S, et al. Among authors: faden ai. Proc Natl Acad Sci U S A. 2005 Jun 7;102(23):8333-8. doi: 10.1073/pnas.0500989102. Epub 2005 May 27. Proc Natl Acad Sci U S A. 2005. PMID: 15923260 Free PMC article.

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