Lawrence BP - Search Results

1

Activation of the aryl hydrocarbon receptor during development enhances the pulmonary CD4+ T-cell response to viral infection.

Boule LA, Winans B, Lambert K, Vorderstrasse BA, Topham DJ, Pavelka MS Jr, Lawrence BP. Boule LA, et al. Among authors: lawrence bp. Am J Physiol Lung Cell Mol Physiol. 2015 Aug 1;309(3):L305-13. doi: 10.1152/ajplung.00135.2015. Epub 2015 Jun 12. Am J Physiol Lung Cell Mol Physiol. 2015. PMID: 26071552 Free PMC article.

2

Examining the relationship between impaired host resistance and altered immune function in mice treated with TCDD.

Vorderstrasse BA, Bohn AA, Lawrence BP. Vorderstrasse BA, et al. Among authors: lawrence bp. Toxicology. 2003 Jun 3;188(1):15-28. doi: 10.1016/s0300-483x(02)00749-7. Toxicology. 2003. PMID: 12748038

3

A novel effect of dioxin: exposure during pregnancy severely impairs mammary gland differentiation.

Vorderstrasse BA, Fenton SE, Bohn AA, Cundiff JA, Lawrence BP. Vorderstrasse BA, et al. Among authors: lawrence bp. Toxicol Sci. 2004 Apr;78(2):248-57. doi: 10.1093/toxsci/kfh062. Epub 2004 Jan 12. Toxicol Sci. 2004. PMID: 14718648

4

Fewer CTL, not enhanced NK cells, are sufficient for viral clearance from the lungs of immunocompromised mice.

Neff-LaFord HD, Vorderstrasse BA, Lawrence BP. Neff-LaFord HD, et al. Among authors: lawrence bp. Cell Immunol. 2003 Nov;226(1):54-64. doi: 10.1016/j.cellimm.2003.11.005. Cell Immunol. 2003. PMID: 14746808

5

Activation of the aryl hydrocarbon receptor diminishes the memory response to homotypic influenza virus infection but does not impair host resistance.

Lawrence BP, Vorderstrasse BA. Lawrence BP, et al. Toxicol Sci. 2004 Jun;79(2):304-14. doi: 10.1093/toxsci/kfh094. Epub 2004 Feb 19. Toxicol Sci. 2004. PMID: 14976337

6

Activation of the aryl hydrocarbon receptor increases pulmonary neutrophilia and diminishes host resistance to influenza A virus.

Teske S, Bohn AA, Regal JF, Neumiller JJ, Lawrence BP. Teske S, et al. Among authors: lawrence bp. Am J Physiol Lung Cell Mol Physiol. 2005 Jul;289(1):L111-24. doi: 10.1152/ajplung.00318.2004. Epub 2005 Mar 25. Am J Physiol Lung Cell Mol Physiol. 2005. PMID: 15792965 Free article.

7

A dose-response study of the effects of prenatal and lactational exposure to TCDD on the immune response to influenza a virus.

Vorderstrasse BA, Cundiff JA, Lawrence BP. Vorderstrasse BA, et al. Among authors: lawrence bp. J Toxicol Environ Health A. 2006 Mar;69(6):445-63. doi: 10.1080/15287390500246985. J Toxicol Environ Health A. 2006. PMID: 16574621

8

Protection against lethal challenge with Streptococcus pneumoniae is conferred by aryl hydrocarbon receptor activation but is not associated with an enhanced inflammatory response.

Vorderstrasse BA, Lawrence BP. Vorderstrasse BA, et al. Among authors: lawrence bp. Infect Immun. 2006 Oct;74(10):5679-86. doi: 10.1128/IAI.00837-06. Infect Immun. 2006. PMID: 16988243 Free PMC article.

9

Aryl hydrocarbon receptor activation impairs the priming but not the recall of influenza virus-specific CD8+ T cells in the lung.

Lawrence BP, Roberts AD, Neumiller JJ, Cundiff JA, Woodland DL. Lawrence BP, et al. J Immunol. 2006 Nov 1;177(9):5819-28. doi: 10.4049/jimmunol.177.9.5819. J Immunol. 2006. PMID: 17056506

10

Activation of the aryl hydrocarbon receptor is essential for mediating the anti-inflammatory effects of a novel low-molecular-weight compound.

Lawrence BP, Denison MS, Novak H, Vorderstrasse BA, Harrer N, Neruda W, Reichel C, Woisetschläger M. Lawrence BP, et al. Blood. 2008 Aug 15;112(4):1158-65. doi: 10.1182/blood-2007-08-109645. Epub 2008 Feb 12. Blood. 2008. PMID: 18270326 Free PMC article.

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