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SAP155-mediated splicing of FUSE-binding protein-interacting repressor serves as a molecular switch for c-myc gene expression.
Matsushita K, Kajiwara T, Tamura M, Satoh M, Tanaka N, Tomonaga T, Matsubara H, Shimada H, Yoshimoto R, Ito A, Kubo S, Natsume T, Levens D, Yoshida M, Nomura F. Matsushita K, et al. Among authors: tanaka n. Mol Cancer Res. 2012 Jun;10(6):787-99. doi: 10.1158/1541-7786.MCR-11-0462. Epub 2012 Apr 11. Mol Cancer Res. 2012. PMID: 22496461 Free PMC article.
Haploinsufficiency of the c-myc transcriptional repressor FIR, as a dominant negative-alternative splicing model, promoted p53-dependent T-cell acute lymphoblastic leukemia progression by activating Notch1.
Matsushita K, Kitamura K, Rahmutulla B, Tanaka N, Ishige T, Satoh M, Hoshino T, Miyagi S, Mori T, Itoga S, Shimada H, Tomonaga T, Kito M, Nakajima-Takagi Y, Kubo S, Nakaseko C, Hatano M, Miki T, Matsuo M, Fukuyo M, Kaneda A, Iwama A, Nomura F. Matsushita K, et al. Among authors: tanaka n. Oncotarget. 2015 Mar 10;6(7):5102-17. doi: 10.18632/oncotarget.3244. Oncotarget. 2015. PMID: 25671302 Free PMC article.
6,566 results