Porse B - Search Results

1

Regulation of Trib2 by an E2F1-C/EBPα feedback loop in AML cell proliferation.

Rishi L, Hannon M, Salomè M, Hasemann M, Frank AK, Campos J, Timoney J, O'Connor C, Cahill MR, Porse B, Keeshan K. Rishi L, et al. Among authors: porse b. Blood. 2014 Apr 10;123(15):2389-400. doi: 10.1182/blood-2013-07-511683. Epub 2014 Feb 10. Blood. 2014. PMID: 24516045 Free article.

2

Loss of C/EBP alpha cell cycle control increases myeloid progenitor proliferation and transforms the neutrophil granulocyte lineage.

Porse BT, Bryder D, Theilgaard-Mönch K, Hasemann MS, Anderson K, Damgaard I, Jacobsen SE, Nerlov C. Porse BT, et al. J Exp Med. 2005 Jul 4;202(1):85-96. doi: 10.1084/jem.20050067. Epub 2005 Jun 27. J Exp Med. 2005. PMID: 15983063 Free PMC article.

3

Mutation of C/EBPalpha predisposes to the development of myeloid leukemia in a retroviral insertional mutagenesis screen.

Hasemann MS, Damgaard I, Schuster MB, Theilgaard-Mönch K, Sørensen AB, Mrsic A, Krugers T, Ylstra B, Pedersen FS, Nerlov C, Porse BT. Hasemann MS, et al. Among authors: porse bt. Blood. 2008 Apr 15;111(8):4309-21. doi: 10.1182/blood-2007-06-097790. Epub 2008 Jan 22. Blood. 2008. PMID: 18212247 Free article.

4

Phosphorylation of serine 248 of C/EBPα is dispensable for myelopoiesis but its disruption leads to a low penetrant myeloid disorder with long latency.

Hasemann MS, Schuster MB, Frank AK, Theilgaard-Mönch K, Pedersen TÅ, Nerlov C, Porse BT. Hasemann MS, et al. Among authors: porse bt. PLoS One. 2012;7(6):e38841. doi: 10.1371/journal.pone.0038841. Epub 2012 Jun 8. PLoS One. 2012. PMID: 22715416 Free PMC article.

5

Lack of the p42 form of C/EBPα leads to spontaneous immortalization and lineage infidelity of committed myeloid progenitors.

Schuster MB, Frank AK, Bagger FO, Rapin N, Vikesaa J, Porse BT. Schuster MB, et al. Among authors: porse bt. Exp Hematol. 2013 Oct;41(10):882-893.e16. doi: 10.1016/j.exphem.2013.06.003. Epub 2013 Jul 2. Exp Hematol. 2013. PMID: 23831605 Free article.

6

Comparing cancer vs normal gene expression profiles identifies new disease entities and common transcriptional programs in AML patients.

Rapin N, Bagger FO, Jendholm J, Mora-Jensen H, Krogh A, Kohlmann A, Thiede C, Borregaard N, Bullinger L, Winther O, Theilgaard-Mönch K, Porse BT. Rapin N, et al. Blood. 2014 Feb 6;123(6):894-904. doi: 10.1182/blood-2013-02-485771. Epub 2013 Dec 20. Blood. 2014. PMID: 24363398 Free article.

7

Initiation of MLL-rearranged AML is dependent on C/EBPα.

Ohlsson E, Hasemann MS, Willer A, Lauridsen FK, Rapin N, Jendholm J, Porse BT. Ohlsson E, et al. J Exp Med. 2014 Jan 13;211(1):5-13. doi: 10.1084/jem.20130932. Epub 2013 Dec 23. J Exp Med. 2014. PMID: 24367003 Free PMC article.

8

Integrative analysis of histone ChIP-seq and transcription data using Bayesian mixture models.

Klein HU, Schäfer M, Porse BT, Hasemann MS, Ickstadt K, Dugas M. Klein HU, et al. Bioinformatics. 2014 Apr 15;30(8):1154-1162. doi: 10.1093/bioinformatics/btu003. Epub 2014 Jan 7. Bioinformatics. 2014. PMID: 24403540

9

C/EBPα is dispensable for the ontogeny of PD-1+ CD4+ memory T cells but restricts their expansion in an age-dependent manner.

Norrie IC, Ohlsson E, Nielsen O, Hasemann MS, Porse BT. Norrie IC, et al. PLoS One. 2014 Jan 3;9(1):e84728. doi: 10.1371/journal.pone.0084728. eCollection 2014. PLoS One. 2014. PMID: 24404186 Free PMC article.

10

C/EBPα is required for long-term self-renewal and lineage priming of hematopoietic stem cells and for the maintenance of epigenetic configurations in multipotent progenitors.

Hasemann MS, Lauridsen FK, Waage J, Jakobsen JS, Frank AK, Schuster MB, Rapin N, Bagger FO, Hoppe PS, Schroeder T, Porse BT. Hasemann MS, et al. Among authors: porse bt. PLoS Genet. 2014 Jan;10(1):e1004079. doi: 10.1371/journal.pgen.1004079. Epub 2014 Jan 9. PLoS Genet. 2014. PMID: 24415956 Free PMC article.

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