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Inhibition of ER stress and unfolding protein response pathways causes skeletal muscle wasting during cancer cachexia.
Bohnert KR, Gallot YS, Sato S, Xiong G, Hindi SM, Kumar A. Bohnert KR, et al. Among authors: sato s. FASEB J. 2016 Sep;30(9):3053-68. doi: 10.1096/fj.201600250RR. Epub 2016 May 20. FASEB J. 2016. PMID: 27206451 Free PMC article.
Our study provides initial evidence that ER stress and UPR pathways are essential for maintaining skeletal muscle mass and strength and for protection against cancer cachexia.-Bohnert, K. R., Gallot, Y. S., Sato, S., Xiong, G., Hindi, S. M., Kumar, A. …
Our study provides initial evidence that ER stress and UPR pathways are essential for maintaining skeletal muscle mass and strength and for …
TAK1 regulates skeletal muscle mass and mitochondrial function.
Hindi SM, Sato S, Xiong G, Bohnert KR, Gibb AA, Gallot YS, McMillan JD, Hill BG, Uchida S, Kumar A. Hindi SM, et al. Among authors: sato s. JCI Insight. 2018 Feb 8;3(3):e98441. doi: 10.1172/jci.insight.98441. eCollection 2018 Feb 8. JCI Insight. 2018. PMID: 29415881 Free PMC article.
Editorial: Emerging Mechanisms for Skeletal Muscle Mass Regulation.
Ogura Y, Sato S, Gallot YS, Arthur ST. Ogura Y, et al. Among authors: sato s. Front Cell Dev Biol. 2021 Sep 27;9:764095. doi: 10.3389/fcell.2021.764095. eCollection 2021. Front Cell Dev Biol. 2021. PMID: 34646832 Free PMC article. No abstract available.
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