Martin GM - Search Results

1

A novel functional low-density lipoprotein receptor-related protein 6 gene alternative splice variant is associated with Alzheimer's disease.

Alarcón MA, Medina MA, Hu Q, Avila ME, Bustos BI, Pérez-Palma E, Peralta A, Salazar P, Ugarte GD, Reyes AE, Martin GM, Opazo C, Moon RT, De Ferrari GV. Alarcón MA, et al. Among authors: martin gm. Neurobiol Aging. 2013 Jun;34(6):1709.e9-18. doi: 10.1016/j.neurobiolaging.2012.11.004. Epub 2012 Dec 6. Neurobiol Aging. 2013. PMID: 23218566

2

A candidate molecular mechanism for the association of an intronic polymorphism of FE65 with resistance to very late onset dementia of the Alzheimer type.

Hu Q, Cool BH, Wang B, Hearn MG, Martin GM. Hu Q, et al. Among authors: martin gm. Hum Mol Genet. 2002 Feb 15;11(4):465-75. doi: 10.1093/hmg/11.4.465. Hum Mol Genet. 2002. PMID: 11854179

3

A flanking gene problem leads to the discovery of a Gprc5b splice variant predominantly expressed in C57Bl/6J mouse brain and in maturing neurons.

Cool BH, Chan GC, Lee L, Oshima J, Martin GM, Hu Q. Cool BH, et al. Among authors: martin gm. PLoS One. 2010 Apr 26;5(4):e10351. doi: 10.1371/journal.pone.0010351. PLoS One. 2010. PMID: 20436672 Free PMC article.

4

Structural and functional characterization of a novel FE65 protein product up-regulated in cognitively impaired FE65 knockout mice.

Cool BH, Zitnik G, Martin GM, Hu Q. Cool BH, et al. Among authors: martin gm. J Neurochem. 2010 Jan;112(2):410-9. doi: 10.1111/j.1471-4159.2009.06456.x. Epub 2009 Oct 27. J Neurochem. 2010. PMID: 19860855 Free PMC article.

5

Alternatively spliced isoforms of FE65 serve as neuron-specific and non-neuronal markers.

Hu Q, Hearn MG, Jin LW, Bressler SL, Martin GM. Hu Q, et al. Among authors: martin gm. J Neurosci Res. 1999 Dec 1;58(5):632-40. doi: 10.1002/(sici)1097-4547(19991201)58:5<632::aid-jnr4>3.0.co;2-p. J Neurosci Res. 1999. PMID: 10561691

6

Broadly altered expression of the mRNA isoforms of FE65, a facilitator of beta amyloidogenesis, in Alzheimer cerebellum and other brain regions.

Hu Q, Jin LW, Starbuck MY, Martin GM. Hu Q, et al. Among authors: martin gm. J Neurosci Res. 2000 Apr 1;60(1):73-86. doi: 10.1002/(SICI)1097-4547(20000401)60:1<73::AID-JNR8>3.0.CO;2-S. J Neurosci Res. 2000. PMID: 10723070

7

Alterations of chaperone protein expression in presenilin mutant neurons in response to glutamate excitotoxicity.

Maezawa I, Wang B, Hu Q, Martin GM, Jin LW, Oshima J. Maezawa I, et al. Among authors: martin gm. Pathol Int. 2002 Sep;52(9):551-4. doi: 10.1046/j.1440-1827.2002.01398.x. Pathol Int. 2002. PMID: 12406183

8

Localizations of endogenous APP/APP-proteolytic products are consistent with microtubular transport.

Zitnik G, Wang L, Martin GM, Hu Q. Zitnik G, et al. Among authors: martin gm. J Mol Neurosci. 2007;31(1):59-68. doi: 10.1007/BF02686118. J Mol Neurosci. 2007. PMID: 17416970

9

Endoproteolytic cleavage of FE65 converts the adaptor protein to a potent suppressor of the sAPPalpha pathway in primates.

Hu Q, Wang L, Yang Z, Cool BH, Zitnik G, Martin GM. Hu Q, et al. Among authors: martin gm. J Biol Chem. 2005 Apr 1;280(13):12548-58. doi: 10.1074/jbc.M411855200. Epub 2005 Jan 12. J Biol Chem. 2005. PMID: 15647266 Free article.

10

A dominant role for FE65 (APBB1) in nuclear signaling.

Yang Z, Cool BH, Martin GM, Hu Q. Yang Z, et al. Among authors: martin gm. J Biol Chem. 2006 Feb 17;281(7):4207-14. doi: 10.1074/jbc.M508445200. Epub 2005 Dec 6. J Biol Chem. 2006. PMID: 16332686 Free article.

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