Farrar EJ - Search Results

1

Valvular heart diseases in the developing world: developmental biology takes center stage.

Farrar EJ, Butcher JT. Farrar EJ, et al. J Heart Valve Dis. 2012 Mar;21(2):234-40. J Heart Valve Dis. 2012. PMID: 22645860 Free PMC article. Review.

2

Notch-Tnf signalling is required for development and homeostasis of arterial valves.

Wang Y, Wu B, Farrar E, Lui W, Lu P, Zhang D, Alfieri CM, Mao K, Chu M, Yang D, Xu D, Rauchman M, Taylor V, Conway SJ, Yutzey KE, Butcher JT, Zhou B. Wang Y, et al. Eur Heart J. 2017 Mar 1;38(9):675-686. doi: 10.1093/eurheartj/ehv520. Eur Heart J. 2017. PMID: 26491108 Free PMC article.

3

NFκB (Nuclear Factor κ-Light-Chain Enhancer of Activated B Cells) Activity Regulates Cell-Type-Specific and Context-Specific Susceptibility to Calcification in the Aortic Valve.

Gee T, Farrar E, Wang Y, Wu B, Hsu K, Zhou B, Butcher J. Gee T, et al. Arterioscler Thromb Vasc Biol. 2020 Mar;40(3):638-655. doi: 10.1161/ATVBAHA.119.313248. Epub 2020 Jan 2. Arterioscler Thromb Vasc Biol. 2020. PMID: 31893948 Free PMC article.

4

The mechanobiology of mitral valve function, degeneration, and repair.

Richards JM, Farrar EJ, Kornreich BG, Moїse NS, Butcher JT. Richards JM, et al. Among authors: farrar ej. J Vet Cardiol. 2012 Mar;14(1):47-58. doi: 10.1016/j.jvc.2012.01.002. Epub 2012 Feb 25. J Vet Cardiol. 2012. PMID: 22366572 Free PMC article. Review.

5

Heterogeneous susceptibility of valve endothelial cells to mesenchymal transformation in response to TNFα.

Farrar EJ, Butcher JT. Farrar EJ, et al. Ann Biomed Eng. 2014 Jan;42(1):149-61. doi: 10.1007/s10439-013-0894-3. Epub 2013 Aug 27. Ann Biomed Eng. 2014. PMID: 23982279 Free PMC article.

6

Endothelial-derived oxidative stress drives myofibroblastic activation and calcification of the aortic valve.

Farrar EJ, Huntley GD, Butcher J. Farrar EJ, et al. PLoS One. 2015 Apr 13;10(4):e0123257. doi: 10.1371/journal.pone.0123257. eCollection 2015. PLoS One. 2015. PMID: 25874717 Free PMC article.

7

Valve interstitial cell tensional homeostasis directs calcification and extracellular matrix remodeling processes via RhoA signaling.

Farrar EJ, Pramil V, Richards JM, Mosher CZ, Butcher JT. Farrar EJ, et al. Biomaterials. 2016 Oct;105:25-37. doi: 10.1016/j.biomaterials.2016.07.034. Epub 2016 Jul 29. Biomaterials. 2016. PMID: 27497058 Free PMC article.

8

OCT4-mediated inflammation induces cell reprogramming at the origin of cardiac valve development and calcification.

Farrar EJ, Hiriart E, Mahmut A, Jagla B, Peal DS, Milan DJ, Butcher JT, Puceat M. Farrar EJ, et al. Sci Adv. 2021 Nov 5;7(45):eabf7910. doi: 10.1126/sciadv.abf7910. Epub 2021 Nov 5. Sci Adv. 2021. PMID: 34739324 Free PMC article.

9

Inflammatory cytokines promote mesenchymal transformation in embryonic and adult valve endothelial cells.

Mahler GJ, Farrar EJ, Butcher JT. Mahler GJ, et al. Among authors: farrar ej. Arterioscler Thromb Vasc Biol. 2013 Jan;33(1):121-30. doi: 10.1161/ATVBAHA.112.300504. Epub 2012 Oct 25. Arterioscler Thromb Vasc Biol. 2013. PMID: 23104848 Free PMC article.

10

Correction: Endothelial-derived oxidative stress drives myofibroblastic activation and calcification of the aortic valve.

Farrar EJ, Huntley GD, Butcher J. Farrar EJ, et al. PLoS One. 2015 May 18;10(5):e0128850. doi: 10.1371/journal.pone.0128850. eCollection 2015. PLoS One. 2015. PMID: 25984758 Free PMC article. No abstract available.

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