Kill IR - Search Results

1

Farnesyltransferase inhibitor treatment restores chromosome territory positions and active chromosome dynamics in Hutchinson-Gilford progeria syndrome cells.

Mehta IS, Eskiw CH, Arican HD, Kill IR, Bridger JM. Mehta IS, et al. Among authors: kill ir. Genome Biol. 2011 Aug 12;12(8):R74. doi: 10.1186/gb-2011-12-8-r74. Genome Biol. 2011. PMID: 21838864 Free PMC article.

2

Towards a Drosophila model of Hutchinson-Gilford progeria syndrome.

Beard GS, Bridger JM, Kill IR, Tree DR. Beard GS, et al. Among authors: kill ir. Biochem Soc Trans. 2008 Dec;36(Pt 6):1389-92. doi: 10.1042/BST0361389. Biochem Soc Trans. 2008. PMID: 19021561

3

Progeria, the nucleolus and farnesyltransferase inhibitors.

Mehta IS, Bridger JM, Kill IR. Mehta IS, et al. Among authors: kill ir. Biochem Soc Trans. 2010 Feb;38(Pt 1):287-91. doi: 10.1042/BST0380287. Biochem Soc Trans. 2010. PMID: 20074076

4

Progeria Research Day at Brunel University.

Bridger JM, Eskiw CH, Makarov EM, Tree D, Kill IR. Bridger JM, et al. Among authors: kill ir. Nucleus. 2011 Nov-Dec;2(6):517-22. doi: 10.4161/nucl.2.6.17605. Epub 2011 Nov 1. Nucleus. 2011. PMID: 22064469 Free PMC article. Review.

5

The non-random repositioning of whole chromosomes and individual gene loci in interphase nuclei and its relevance in disease, infection, aging, and cancer.

Bridger JM, Arican-Gotkas HD, Foster HA, Godwin LS, Harvey A, Kill IR, Knight M, Mehta IS, Ahmed MH. Bridger JM, et al. Among authors: kill ir. Adv Exp Med Biol. 2014;773:263-79. doi: 10.1007/978-1-4899-8032-8_12. Adv Exp Med Biol. 2014. PMID: 24563352 Review.

6

Farnesyltransferase inhibitor and rapamycin correct aberrant genome organisation and decrease DNA damage respectively, in Hutchinson-Gilford progeria syndrome fibroblasts.

Bikkul MU, Clements CS, Godwin LS, Goldberg MW, Kill IR, Bridger JM. Bikkul MU, et al. Among authors: kill ir. Biogerontology. 2018 Dec;19(6):579-602. doi: 10.1007/s10522-018-9758-4. Epub 2018 Jun 15. Biogerontology. 2018. PMID: 29907918 Free PMC article.

7

Aging of Hutchinson-Gilford progeria syndrome fibroblasts is characterised by hyperproliferation and increased apoptosis.

Bridger JM, Kill IR. Bridger JM, et al. Among authors: kill ir. Exp Gerontol. 2004 May;39(5):717-24. doi: 10.1016/j.exger.2004.02.002. Exp Gerontol. 2004. PMID: 15130666

8

Primary laminopathy fibroblasts display altered genome organization and apoptosis.

Meaburn KJ, Cabuy E, Bonne G, Levy N, Morris GE, Novelli G, Kill IR, Bridger JM. Meaburn KJ, et al. Among authors: kill ir. Aging Cell. 2007 Apr;6(2):139-53. doi: 10.1111/j.1474-9726.2007.00270.x. Epub 2007 Feb 5. Aging Cell. 2007. PMID: 17274801 Free article.

9

Alterations to nuclear architecture and genome behavior in senescent cells.

Mehta IS, Figgitt M, Clements CS, Kill IR, Bridger JM. Mehta IS, et al. Among authors: kill ir. Ann N Y Acad Sci. 2007 Apr;1100:250-63. doi: 10.1196/annals.1395.027. Ann N Y Acad Sci. 2007. PMID: 17460187 Free article. Review.

10

The nuclear lamina. Both a structural framework and a platform for genome organization.

Bridger JM, Foeger N, Kill IR, Herrmann H. Bridger JM, et al. Among authors: kill ir. FEBS J. 2007 Mar;274(6):1354-61. doi: 10.1111/j.1742-4658.2007.05694.x. FEBS J. 2007. PMID: 17489093 Free article. Review.

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