Fröhlich LF - Search Results

1

Modifying the protease, antiprotease pattern by elafin overexpression protects mice from colitis.

Motta JP, Magne L, Descamps D, Rolland C, Squarzoni-Dale C, Rousset P, Martin L, Cenac N, Balloy V, Huerre M, Fröhlich LF, Jenne D, Wartelle J, Belaaouaj A, Mas E, Vinel JP, Alric L, Chignard M, Vergnolle N, Sallenave JM. Motta JP, et al. Among authors: frohlich lf. Gastroenterology. 2011 Apr;140(4):1272-82. doi: 10.1053/j.gastro.2010.12.050. Epub 2011 Jan 1. Gastroenterology. 2011. PMID: 21199654

2

Proteinase 3 and neutrophil elastase enhance inflammation in mice by inactivating antiinflammatory progranulin.

Kessenbrock K, Fröhlich L, Sixt M, Lämmermann T, Pfister H, Bateman A, Belaaouaj A, Ring J, Ollert M, Fässler R, Jenne DE. Kessenbrock K, et al. J Clin Invest. 2008 Jul;118(7):2438-47. doi: 10.1172/JCI34694. J Clin Invest. 2008. PMID: 18568075 Free PMC article.

3

Antineutrophil cytoplasmic autoantibodies against the murine homolog of proteinase 3 (Wegener autoantigen) are pathogenic in vivo.

Pfister H, Ollert M, Fröhlich LF, Quintanilla-Martinez L, Colby TV, Specks U, Jenne DE. Pfister H, et al. Among authors: frohlich lf. Blood. 2004 Sep 1;104(5):1411-8. doi: 10.1182/blood-2004-01-0267. Epub 2004 May 18. Blood. 2004. PMID: 15150076 Free article.

4

Cloning and functional expression of the murine homologue of proteinase 3: implications for the design of murine models of vasculitis.

Jenne DE, Fröhlich L, Hummel AM, Specks U. Jenne DE, et al. FEBS Lett. 1997 May 19;408(2):187-90. doi: 10.1016/s0014-5793(97)00418-3. FEBS Lett. 1997. PMID: 9187364 Free article.

5

Skin wounds and severed nerves heal normally in mice lacking tenascin-C.

Forsberg E, Hirsch E, Fröhlich L, Meyer M, Ekblom P, Aszodi A, Werner S, Fässler R. Forsberg E, et al. Proc Natl Acad Sci U S A. 1996 Jun 25;93(13):6594-9. doi: 10.1073/pnas.93.13.6594. Proc Natl Acad Sci U S A. 1996. PMID: 8692862 Free PMC article.

6

Mapping of a defined neurocan binding site to distinct domains of tenascin-C.

Rauch U, Clement A, Retzler C, Fröhlich L, Fässler R, Göhring W, Faissner A. Rauch U, et al. J Biol Chem. 1997 Oct 24;272(43):26905-12. doi: 10.1074/jbc.272.43.26905. J Biol Chem. 1997. PMID: 9341124 Free article.

7

Genomic structure and refined chromosomal localization of the mouse Ptch2 gene.

Fröhlich L, Liu Z, Beier DR, Lanske B. Fröhlich L, et al. Cytogenet Genome Res. 2002;97(1-2):106-10. doi: 10.1159/000064064. Cytogenet Genome Res. 2002. PMID: 12438747

8

Autosomal dominant pseudohypoparathyroidism type Ib is associated with a heterozygous microdeletion that likely disrupts a putative imprinting control element of GNAS.

Bastepe M, Fröhlich LF, Hendy GN, Indridason OS, Josse RG, Koshiyama H, Körkkö J, Nakamoto JM, Rosenbloom AL, Slyper AH, Sugimoto T, Tsatsoulis A, Crawford JD, Jüppner H. Bastepe M, et al. Among authors: frohlich lf. J Clin Invest. 2003 Oct;112(8):1255-63. doi: 10.1172/JCI19159. J Clin Invest. 2003. PMID: 14561710 Free PMC article.

9

Deletion of the NESP55 differentially methylated region causes loss of maternal GNAS imprints and pseudohypoparathyroidism type Ib.

Bastepe M, Fröhlich LF, Linglart A, Abu-Zahra HS, Tojo K, Ward LM, Jüppner H. Bastepe M, et al. Among authors: frohlich lf. Nat Genet. 2005 Jan;37(1):25-7. doi: 10.1038/ng1487. Epub 2004 Dec 12. Nat Genet. 2005. PMID: 15592469

10

Autosomal-dominant pseudohypoparathyroidism type Ib is caused by different microdeletions within or upstream of the GNAS locus.

Jüppner H, Linglart A, Fröhlich LF, Bastepe M. Jüppner H, et al. Among authors: frohlich lf. Ann N Y Acad Sci. 2006 Apr;1068:250-5. doi: 10.1196/annals.1346.029. Ann N Y Acad Sci. 2006. PMID: 16831926

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