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Page 1
Effects of p38 mitogen-activated protein kinase inhibition on anti-neutrophil cytoplasmic autoantibody pathogenicity in vitro and in vivo.
van der Veen BS, Chen M, Müller R, van Timmeren MM, Petersen AH, Lee PA, Satchell SC, Mathieson PW, Saleem MA, Stegeman CA, Zwerina J, Molema G, Heeringa P. van der Veen BS, et al. Among authors: molema g. Ann Rheum Dis. 2011 Feb;70(2):356-65. doi: 10.1136/ard.2010.129106. Epub 2010 Nov 9. Ann Rheum Dis. 2011. PMID: 21062851
Differential effects of NF-{kappa}B and p38 MAPK inhibitors and combinations thereof on TNF-{alpha}- and IL-1{beta}-induced proinflammatory status of endothelial cells in vitro.
Kuldo JM, Westra J, Asgeirsdóttir SA, Kok RJ, Oosterhuis K, Rots MG, Schouten JP, Limburg PC, Molema G. Kuldo JM, et al. Among authors: molema g. Am J Physiol Cell Physiol. 2005 Nov;289(5):C1229-39. doi: 10.1152/ajpcell.00620.2004. Epub 2005 Jun 22. Am J Physiol Cell Physiol. 2005. PMID: 15972838 Free article.
Site-specific inhibition of glomerulonephritis progression by targeted delivery of dexamethasone to glomerular endothelium.
Asgeirsdóttir SA, Kamps JA, Bakker HI, Zwiers PJ, Heeringa P, van der Weide K, van Goor H, Petersen AH, Morselt H, Moorlag HE, Steenbergen E, Kallenberg CG, Molema G. Asgeirsdóttir SA, et al. Among authors: molema g. Mol Pharmacol. 2007 Jul;72(1):121-31. doi: 10.1124/mol.107.034140. Epub 2007 Apr 23. Mol Pharmacol. 2007. PMID: 17452496
Shock-induced stress induces loss of microvascular endothelial Tie2 in the kidney which is not associated with reduced glomerular barrier function.
van Meurs M, Kurniati NF, Wulfert FM, Asgeirsdottir SA, de Graaf IA, Satchell SC, Mathieson PW, Jongman RM, Kümpers P, Zijlstra JG, Heeringa P, Molema G. van Meurs M, et al. Among authors: molema g. Am J Physiol Renal Physiol. 2009 Aug;297(2):F272-81. doi: 10.1152/ajprenal.00137.2009. Epub 2009 Jun 10. Am J Physiol Renal Physiol. 2009. PMID: 19515812 Free article.
Hemorrhagic shock-induced endothelial cell activation in a spontaneous breathing and a mechanical ventilation hemorrhagic shock model is induced by a proinflammatory response and not by hypoxia.
van Meurs M, Wulfert FM, Jongman RM, Schipper M, Houwertjes MC, Vaneker M, Scheffer GJ, Teppema LJ, Aarts LP, Heeringa P, Zijlstra JG, Molema G. van Meurs M, et al. Among authors: molema g. Anesthesiology. 2011 Sep;115(3):474-82. doi: 10.1097/ALN.0b013e318229a640. Anesthesiology. 2011. PMID: 21778876
MicroRNA-126 contributes to renal microvascular heterogeneity of VCAM-1 protein expression in acute inflammation.
Asgeirsdóttir SA, van Solingen C, Kurniati NF, Zwiers PJ, Heeringa P, van Meurs M, Satchell SC, Saleem MA, Mathieson PW, Banas B, Kamps JA, Rabelink TJ, van Zonneveld AJ, Molema G. Asgeirsdóttir SA, et al. Among authors: molema g. Am J Physiol Renal Physiol. 2012 Jun 15;302(12):F1630-9. doi: 10.1152/ajprenal.00400.2011. Epub 2012 Mar 14. Am J Physiol Renal Physiol. 2012. PMID: 22419694 Free article.
We previously showed that during hemorrhagic shock, VCAM-1 protein was expressed primarily in extraglomerular compartments of the kidney, while E-selectin protein was highly induced in glomeruli only (van Meurs M, Wulfert FM, Knol AJ, de Haes A, Houwertjes M, Aarts LPHJ, Molem
We previously showed that during hemorrhagic shock, VCAM-1 protein was expressed primarily in extraglomerular compartments of the kidney, wh …
210 results