Katayama Y - Search Results

1

Early cellular swelling in experimental traumatic brain injury: a phenomenon mediated by excitatory amino acids.

Katayama Y, Becker DP, Tamura T, Ikezaki K. Katayama Y, et al. Acta Neurochir Suppl (Wien). 1990;51:271-3. doi: 10.1007/978-3-7091-9115-6_92. Acta Neurochir Suppl (Wien). 1990. PMID: 2089915

2

Inhibition of rapid potassium flux during cerebral ischemia in vivo with an excitatory amino acid antagonist.

Katayama Y, Tamura T, Becker DP, Tsubokawa T. Katayama Y, et al. Brain Res. 1991 Dec 24;568(1-2):294-8. doi: 10.1016/0006-8993(91)91412-t. Brain Res. 1991. PMID: 1687669

3

Massive increases in extracellular potassium and the indiscriminate release of glutamate following concussive brain injury.

Katayama Y, Becker DP, Tamura T, Hovda DA. Katayama Y, et al. J Neurosurg. 1990 Dec;73(6):889-900. doi: 10.3171/jns.1990.73.6.0889. J Neurosurg. 1990. PMID: 1977896 Review.

4

Cellular swelling during cerebral ischaemia demonstrated by microdialysis in vivo: preliminary data indicating the role of excitatory amino acids.

Katayama Y, Becker DP, Tamura T, Tsubokawa T. Katayama Y, et al. Acta Neurochir Suppl (Wien). 1990;51:183-5. doi: 10.1007/978-3-7091-9115-6_62. Acta Neurochir Suppl (Wien). 1990. PMID: 2089891

5

The increase in local cerebral glucose utilization following fluid percussion brain injury is prevented with kynurenic acid and is associated with an increase in calcium.

Hovda DA, Yoshino A, Kawamata T, Katayama Y, Fineman I, Becker DP. Hovda DA, et al. Among authors: katayama y. Acta Neurochir Suppl (Wien). 1990;51:331-3. doi: 10.1007/978-3-7091-9115-6_112. Acta Neurochir Suppl (Wien). 1990. PMID: 2089930

6

Lactate accumulation following concussive brain injury: the role of ionic fluxes induced by excitatory amino acids.

Kawamata T, Katayama Y, Hovda DA, Yoshino A, Becker DP. Kawamata T, et al. Among authors: katayama y. Brain Res. 1995 Mar 20;674(2):196-204. doi: 10.1016/0006-8993(94)01444-m. Brain Res. 1995. PMID: 7540925

7

Early cellular swelling during cerebral ischemia in vivo is mediated by excitatory amino acids released from nerve terminals.

Katayama Y, Tamura T, Becker DP, Tsubokawa T. Katayama Y, et al. Brain Res. 1992 Apr 10;577(1):121-6. doi: 10.1016/0006-8993(92)90544-j. Brain Res. 1992. PMID: 1325856

8

Administration of excitatory amino acid antagonists via microdialysis attenuates the increase in glucose utilization seen following concussive brain injury.

Kawamata T, Katayama Y, Hovda DA, Yoshino A, Becker DP. Kawamata T, et al. Among authors: katayama y. J Cereb Blood Flow Metab. 1992 Jan;12(1):12-24. doi: 10.1038/jcbfm.1992.3. J Cereb Blood Flow Metab. 1992. PMID: 1345756

9

Secondary injury and acidosis.

Hovda DA, Becker DP, Katayama Y. Hovda DA, et al. Among authors: katayama y. J Neurotrauma. 1992 Mar;9 Suppl 1:S47-60. J Neurotrauma. 1992. PMID: 1588632 Review.

10

Diffuse prolonged depression of cerebral oxidative metabolism following concussive brain injury in the rat: a cytochrome oxidase histochemistry study.

Hovda DA, Yoshino A, Kawamata T, Katayama Y, Becker DP. Hovda DA, et al. Among authors: katayama y. Brain Res. 1991 Dec 13;567(1):1-10. doi: 10.1016/0006-8993(91)91429-5. Brain Res. 1991. PMID: 1667742

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