Gale RE - Search Results

1

The impact on outcome of the addition of all-trans retinoic acid to intensive chemotherapy in younger patients with nonacute promyelocytic acute myeloid leukemia: overall results and results in genotypic subgroups defined by mutations in NPM1, FLT3, and CEBPA.

Burnett AK, Hills RK, Green C, Jenkinson S, Koo K, Patel Y, Guy C, Gilkes A, Milligan DW, Goldstone AH, Prentice AG, Wheatley K, Linch DC, Gale RE. Burnett AK, et al. Among authors: gale re. Blood. 2010 Feb 4;115(5):948-56. doi: 10.1182/blood-2009-08-236588. Epub 2009 Nov 12. Blood. 2010. PMID: 19965647 Free article. Clinical Trial.

2

Assessment of X-chromosome inactivation patterns using the hypervariable probe M27 beta in normal hemopoietic cells and acute myeloid leukemic blasts.

Gale RE, Wheadon H, Linch DC. Gale RE, et al. Leukemia. 1992 Jul;6(7):649-55. Leukemia. 1992. PMID: 1352560

3

X-chromosome inactivation patterns using HPRT and PGK polymorphisms in haematologically normal and post-chemotherapy females.

Gale RE, Wheadon H, Linch DC. Gale RE, et al. Br J Haematol. 1991 Oct;79(2):193-7. doi: 10.1111/j.1365-2141.1991.tb04521.x. Br J Haematol. 1991. PMID: 1958476

4

Analysis of mutations in the GM-CSF receptor alpha coding sequence in patients with acute myeloid leukaemia and haematologically normal individuals by RT-PCR-SSCP.

Wagner HM, Gale RE, Freeburn RW, Devereux S, Linch DC. Wagner HM, et al. Among authors: gale re. Leukemia. 1994 Sep;8(9):1527-32. Leukemia. 1994. PMID: 7522290

5

Interpretation of X-chromosome inactivation patterns.

Gale RE, Linch DC. Gale RE, et al. Blood. 1994 Oct 1;84(7):2376-8. Blood. 1994. PMID: 7919357 Free article. No abstract available.

6

Lineage involvement in chronic myeloproliferative disorders.

Gale RE, Linch DC. Gale RE, et al. Br J Haematol. 1994 Aug;87(4):882-3. doi: 10.1111/j.1365-2141.1994.tb06763.x. Br J Haematol. 1994. PMID: 7986739 No abstract available.

7

Frequency of clonal remission in acute myeloid leukaemia.

Gale RE, Wheadon H, Goldstone AH, Burnett AK, Linch DC. Gale RE, et al. Lancet. 1993 Jan 16;341(8838):138-42. doi: 10.1016/0140-6736(93)90004-z. Lancet. 1993. PMID: 8093744

8

Tissue specificity of X-chromosome inactivation patterns.

Gale RE, Wheadon H, Boulos P, Linch DC. Gale RE, et al. Blood. 1994 May 15;83(10):2899-905. Blood. 1994. PMID: 8180385 Free article.

9

Investigation of methylation at Hha I sites using the hypervariable probe M27 beta allows improved clonal analysis in myeloid leukaemia and demonstrates differences in methylation between leukaemic and remission samples.

Gale RE, Linch DC. Gale RE, et al. Leukemia. 1994 Jan;8(1):190-4. Leukemia. 1994. PMID: 8289487

10

The beta subunit common to the GM-CSF, IL-3 and IL-5 receptors is highly polymorphic but pathogenic point mutations in patients with acute myeloid leukaemia (AML) are rare.

Freeburn RW, Gale RE, Wagner HM, Linch DC. Freeburn RW, et al. Among authors: gale re. Leukemia. 1996 Jan;10(1):123-9. Leukemia. 1996. PMID: 8558916

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