Binah O - Search Results

1

Functional and developmental properties of human embryonic stem cells-derived cardiomyocytes.

Binah O, Dolnikov K, Sadan O, Shilkrut M, Zeevi-Levin N, Amit M, Danon A, Itskovitz-Eldor J. Binah O, et al. J Electrocardiol. 2007 Nov-Dec;40(6 Suppl):S192-6. doi: 10.1016/j.jelectrocard.2007.05.035. J Electrocardiol. 2007. PMID: 17993321

2

Experimental iron deficiency in rats: mechanical and electrophysiological alterations in the cardiac muscle.

Goldstein D, Felzen B, Youdim M, Lotan R, Binah O. Goldstein D, et al. Among authors: binah o. Clin Sci (Lond). 1996 Aug;91(2):233-9. doi: 10.1042/cs0910233. Clin Sci (Lond). 1996. PMID: 8795449

3

Fas (CD95/Apo-1)-mediated damage to ventricular myocytes induced by cytotoxic T lymphocytes from perforin-deficient mice: a major role for inositol 1,4,5-trisphosphate.

Felzen B, Shilkrut M, Less H, Sarapov I, Maor G, Coleman R, Robinson RB, Berke G, Binah O. Felzen B, et al. Among authors: binah o. Circ Res. 1998 Mar 9;82(4):438-50. doi: 10.1161/01.res.82.4.438. Circ Res. 1998. PMID: 9506704

4

Cardiac dysfunction in murine autoimmune myocarditis.

Less H, Shilkrut M, Rubinstein I, Berke G, Binah O. Less H, et al. Among authors: binah o. J Autoimmun. 1999 May;12(3):209-20. doi: 10.1006/jaut.1998.0273. J Autoimmun. 1999. PMID: 10222030

5

Electrophysiologic perturbations and arrhythmogenic activity caused by activation of the Fas receptor in murine ventricular myocytes: role of the inositol trisphosphate pathway.

Shilkrut M, Gealekman O, Rosen D, Berke G, Woodcock E, Binah O. Shilkrut M, et al. Among authors: binah o. J Cardiovasc Electrophysiol. 2001 Feb;12(2):185-95. doi: 10.1046/j.1540-8167.2001.00185.x. J Cardiovasc Electrophysiol. 2001. PMID: 11232618

6

Human embryonic stem cells can differentiate into myocytes with structural and functional properties of cardiomyocytes.

Kehat I, Kenyagin-Karsenti D, Snir M, Segev H, Amit M, Gepstein A, Livne E, Binah O, Itskovitz-Eldor J, Gepstein L. Kehat I, et al. Among authors: binah o. J Clin Invest. 2001 Aug;108(3):407-14. doi: 10.1172/JCI12131. J Clin Invest. 2001. PMID: 11489934 Free PMC article.

7

Hypoxia predisposes neonatal rat ventricular myocytes to apoptosis induced by activation of the Fas (CD95/Apo-1) receptor: Fas activation and apoptosis in hypoxic myocytes.

Yaniv G, Shilkrut M, Lotan R, Berke G, Larisch S, Binah O. Yaniv G, et al. Among authors: binah o. Cardiovasc Res. 2002 Jun;54(3):611-23. doi: 10.1016/s0008-6363(02)00264-x. Cardiovasc Res. 2002. PMID: 12031707

8

Genetically determined heterogeneity in hemoglobin scavenging and susceptibility to diabetic cardiovascular disease.

Asleh R, Marsh S, Shilkrut M, Binah O, Guetta J, Lejbkowicz F, Enav B, Shehadeh N, Kanter Y, Lache O, Cohen O, Levy NS, Levy AP. Asleh R, et al. Among authors: binah o. Circ Res. 2003 Jun 13;92(11):1193-200. doi: 10.1161/01.RES.0000076889.23082.F1. Epub 2003 May 15. Circ Res. 2003. PMID: 12750308

9

Tyrosine kinases inhibitors block Fas-mediated deleterious effects in normoxic and hypoxic ventricular myocytes.

Shilkrut M, Yaniv G, Asleh R, Levy AP, Larisch S, Binah O. Shilkrut M, et al. Among authors: binah o. J Mol Cell Cardiol. 2003 Oct;35(10):1229-40. doi: 10.1016/s0022-2828(03)00232-3. J Mol Cell Cardiol. 2003. PMID: 14519433

10

The Fas receptor-1,4,5-IP3 cascade: a potential target for treating heart failure and arrhythmias.

Binah O, Shilkrut M, Yaniv G, Larisch S. Binah O, et al. Ann N Y Acad Sci. 2004 May;1015:338-50. doi: 10.1196/annals.1302.030. Ann N Y Acad Sci. 2004. PMID: 15201173

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