Kay GW - Search Results

1

Metabolomic investigation of CLN6 neuronal ceroid lipofuscinosis in affected South Hampshire sheep.

Pears MR, Salek RM, Palmer DN, Kay GW, Mortishire-Smith RJ, Griffin JL. Pears MR, et al. Among authors: kay gw. J Neurosci Res. 2007 Nov 15;85(15):3494-504. doi: 10.1002/jnr.21343. J Neurosci Res. 2007. PMID: 17510975

2

Aggregation chimeras provide evidence of in vivo intercellular correction in ovine CLN6 neuronal ceroid lipofuscinosis (Batten disease).

Barry LA, Kay GW, Mitchell NL, Murray SJ, Jay NP, Palmer DN. Barry LA, et al. Among authors: kay gw. PLoS One. 2022 Apr 11;17(4):e0261544. doi: 10.1371/journal.pone.0261544. eCollection 2022. PLoS One. 2022. PMID: 35404973 Free PMC article.

3

Glial activation spreads from specific cerebral foci and precedes neurodegeneration in presymptomatic ovine neuronal ceroid lipofuscinosis (CLN6).

Oswald MJ, Palmer DN, Kay GW, Shemilt SJ, Rezaie P, Cooper JD. Oswald MJ, et al. Among authors: kay gw. Neurobiol Dis. 2005 Oct;20(1):49-63. doi: 10.1016/j.nbd.2005.01.025. Neurobiol Dis. 2005. PMID: 16137566

4

Activation of non-neuronal cells within the prenatal developing brain of sheep with neuronal ceroid lipofuscinosis.

Kay GW, Palmer DN, Rezaie P, Cooper JD. Kay GW, et al. Brain Pathol. 2006 Apr;16(2):110-6. doi: 10.1111/j.1750-3639.2006.00002.x. Brain Pathol. 2006. PMID: 16768750 Free PMC article.

5

A missense mutation (c.184C>T) in ovine CLN6 causes neuronal ceroid lipofuscinosis in Merino sheep whereas affected South Hampshire sheep have reduced levels of CLN6 mRNA.

Tammen I, Houweling PJ, Frugier T, Mitchell NL, Kay GW, Cavanagh JA, Cook RW, Raadsma HW, Palmer DN. Tammen I, et al. Among authors: kay gw. Biochim Biophys Acta. 2006 Oct;1762(10):898-905. doi: 10.1016/j.bbadis.2006.09.004. Epub 2006 Sep 12. Biochim Biophys Acta. 2006. PMID: 17046213 Free article.

6

Location and connectivity determine GABAergic interneuron survival in the brains of South Hampshire sheep with CLN6 neuronal ceroid lipofuscinosis.

Oswald MJ, Palmer DN, Kay GW, Barwell KJ, Cooper JD. Oswald MJ, et al. Among authors: kay gw. Neurobiol Dis. 2008 Oct;32(1):50-65. doi: 10.1016/j.nbd.2008.06.004. Epub 2008 Jun 25. Neurobiol Dis. 2008. PMID: 18634879 Free PMC article.

7

Neuropeptide changes and neuroactive amino acids in CSF from humans and sheep with neuronal ceroid lipofuscinoses (NCLs, Batten disease).

Kay GW, Verbeek MM, Furlong JM, Willemsen MA, Palmer DN. Kay GW, et al. Neurochem Int. 2009 Dec;55(8):783-8. doi: 10.1016/j.neuint.2009.07.012. Epub 2009 Aug 5. Neurochem Int. 2009. PMID: 19664668 Free PMC article.

8

The specific loss of GnRH-positive neurons from the hypothalamus of sheep with CLN6 neuronal ceroid lipofuscinosis occurs without glial activation and has only minor effects on reproduction.

Kay GW, Jay NP, Palmer DN. Kay GW, et al. Neurobiol Dis. 2011 Mar;41(3):614-23. doi: 10.1016/j.nbd.2010.11.008. Epub 2010 Nov 24. Neurobiol Dis. 2011. PMID: 21111820

9

Lentiviral-mediated gene transfer to the sheep brain: implications for gene therapy in Batten disease.

Linterman KS, Palmer DN, Kay GW, Barry LA, Mitchell NL, McFarlane RG, Black MA, Sands MS, Hughes SM. Linterman KS, et al. Among authors: kay gw. Hum Gene Ther. 2011 Aug;22(8):1011-20. doi: 10.1089/hum.2011.026. Epub 2011 May 19. Hum Gene Ther. 2011. PMID: 21595499 Free PMC article.

10

Chronic oral administration of minocycline to sheep with ovine CLN6 neuronal ceroid lipofuscinosis maintains pharmacological concentrations in the brain but does not suppress neuroinflammation or disease progression.

Kay GW, Palmer DN. Kay GW, et al. J Neuroinflammation. 2013 Jul 30;10:97. doi: 10.1186/1742-2094-10-97. J Neuroinflammation. 2013. PMID: 23899308 Free PMC article.

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