Wang M - Search Results

1

The A subunit of type IIb enterotoxin (LT-IIb) suppresses the proinflammatory potential of the B subunit and its ability to recruit and interact with TLR2.

Liang S, Wang M, Triantafilou K, Triantafilou M, Nawar HF, Russell MW, Connell TD, Hajishengallis G. Liang S, et al. Among authors: wang m. J Immunol. 2007 Apr 15;178(8):4811-9. doi: 10.4049/jimmunol.178.8.4811. J Immunol. 2007. PMID: 17404262

2

Porphyromonas gingivalis fimbriae proactively modulate beta2 integrin adhesive activity and promote binding to and internalization by macrophages.

Hajishengallis G, Wang M, Harokopakis E, Triantafilou M, Triantafilou K. Hajishengallis G, et al. Among authors: wang m. Infect Immun. 2006 Oct;74(10):5658-66. doi: 10.1128/IAI.00784-06. Infect Immun. 2006. PMID: 16988241 Free PMC article.

3

Ganglioside GD1a is an essential coreceptor for Toll-like receptor 2 signaling in response to the B subunit of type IIb enterotoxin.

Liang S, Wang M, Tapping RI, Stepensky V, Nawar HF, Triantafilou M, Triantafilou K, Connell TD, Hajishengallis G. Liang S, et al. Among authors: wang m. J Biol Chem. 2007 Mar 9;282(10):7532-42. doi: 10.1074/jbc.M611722200. Epub 2007 Jan 16. J Biol Chem. 2007. PMID: 17227759 Free article.

4

Fimbrial proteins of porphyromonas gingivalis mediate in vivo virulence and exploit TLR2 and complement receptor 3 to persist in macrophages.

Wang M, Shakhatreh MA, James D, Liang S, Nishiyama S, Yoshimura F, Demuth DR, Hajishengallis G. Wang M, et al. J Immunol. 2007 Aug 15;179(4):2349-58. doi: 10.4049/jimmunol.179.4.2349. J Immunol. 2007. PMID: 17675496

5

Complement receptor 3 blockade promotes IL-12-mediated clearance of Porphyromonas gingivalis and negates its virulence in vivo.

Hajishengallis G, Shakhatreh MA, Wang M, Liang S. Hajishengallis G, et al. Among authors: wang m. J Immunol. 2007 Aug 15;179(4):2359-67. doi: 10.4049/jimmunol.179.4.2359. J Immunol. 2007. PMID: 17675497

6

Lipid raft-dependent uptake, signalling and intracellular fate of Porphyromonas gingivalis in mouse macrophages.

Wang M, Hajishengallis G. Wang M, et al. Cell Microbiol. 2008 Oct;10(10):2029-42. doi: 10.1111/j.1462-5822.2008.01185.x. Epub 2008 Jun 10. Cell Microbiol. 2008. PMID: 18547335 Free PMC article.

7

Pathogen induction of CXCR4/TLR2 cross-talk impairs host defense function.

Hajishengallis G, Wang M, Liang S, Triantafilou M, Triantafilou K. Hajishengallis G, et al. Among authors: wang m. Proc Natl Acad Sci U S A. 2008 Sep 9;105(36):13532-7. doi: 10.1073/pnas.0803852105. Epub 2008 Sep 2. Proc Natl Acad Sci U S A. 2008. PMID: 18765807 Free PMC article.

8

Importance of TLR2 in early innate immune response to acute pulmonary infection with Porphyromonas gingivalis in mice.

Hajishengallis G, Wang M, Bagby GJ, Nelson S. Hajishengallis G, et al. Among authors: wang m. J Immunol. 2008 Sep 15;181(6):4141-9. doi: 10.4049/jimmunol.181.6.4141. J Immunol. 2008. PMID: 18768871 Free PMC article.

9

Subversion of innate immunity by periodontopathic bacteria via exploitation of complement receptor-3.

Hajishengallis G, Wang M, Liang S, Shakhatreh MA, James D, Nishiyama S, Yoshimura F, Demuth DR. Hajishengallis G, et al. Among authors: wang m. Adv Exp Med Biol. 2008;632:203-19. doi: 10.1007/978-0-387-78952-1_15. Adv Exp Med Biol. 2008. PMID: 19025124 Free PMC article. Review.

10

Induction of distinct TLR2-mediated proinflammatory and proadhesive signaling pathways in response to Porphyromonas gingivalis fimbriae.

Hajishengallis G, Wang M, Liang S. Hajishengallis G, et al. Among authors: wang m. J Immunol. 2009 Jun 1;182(11):6690-6. doi: 10.4049/jimmunol.0900524. J Immunol. 2009. PMID: 19454663 Free PMC article.

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