Bonventre JV - Search Results

1

Renal stem cells in recovery from acute kidney injury.

Humphreys BD, Duffield JS, Bonventre JV. Humphreys BD, et al. Among authors: bonventre jv. Minerva Urol Nefrol. 2006 Dec;58(4):329-37. Minerva Urol Nefrol. 2006. PMID: 17268398 Review.

2

Dedifferentiation and proliferation of surviving epithelial cells in acute renal failure.

Bonventre JV. Bonventre JV. J Am Soc Nephrol. 2003 Jun;14 Suppl 1:S55-61. doi: 10.1097/01.asn.0000067652.51441.21. J Am Soc Nephrol. 2003. PMID: 12761240 Review.

3

Recent advances in the pathophysiology of ischemic acute renal failure.

Bonventre JV, Weinberg JM. Bonventre JV, et al. J Am Soc Nephrol. 2003 Aug;14(8):2199-210. doi: 10.1097/01.asn.0000079785.13922.f6. J Am Soc Nephrol. 2003. PMID: 12874476 Review. No abstract available.

4

Molecular response to cytotoxic injury: role of inflammation, MAP kinases, and endoplasmic reticulum stress response.

Bonventre JV. Bonventre JV. Semin Nephrol. 2003 Sep;23(5):439-48. doi: 10.1016/s0270-9295(03)00115-3. Semin Nephrol. 2003. PMID: 13680533 Review.

5

Dose of dialysis in acute renal failure.

Luyckx VA, Bonventre JV. Luyckx VA, et al. Among authors: bonventre jv. Semin Dial. 2004 Jan-Feb;17(1):30-6. doi: 10.1111/j.1525-139x.2004.17111.x. Semin Dial. 2004. PMID: 14717809 Review.

6

Ischemic acute renal failure: an inflammatory disease?

Bonventre JV, Zuk A. Bonventre JV, et al. Kidney Int. 2004 Aug;66(2):480-5. doi: 10.1111/j.1523-1755.2004.761_2.x. Kidney Int. 2004. PMID: 15253693 Free article. Review.

7

Pathophysiology of ischemic acute renal failure. Inflammation, lung-kidney cross-talk, and biomarkers.

Bonventre JV. Bonventre JV. Contrib Nephrol. 2004;144:19-30. Contrib Nephrol. 2004. PMID: 15264395 Review. No abstract available.

8

Biologic markers for the early detection of acute kidney injury.

Han WK, Bonventre JV. Han WK, et al. Among authors: bonventre jv. Curr Opin Crit Care. 2004 Dec;10(6):476-82. doi: 10.1097/01.ccx.0000145095.90327.f2. Curr Opin Crit Care. 2004. PMID: 15616389 Review.

9

cPLA2-interacting protein, PLIP, causes apoptosis and decreases G1 phase in mesangial cells.

Muckova K, Duffield JS, Held KD, Bonventre JV, Sheridan AM. Muckova K, et al. Among authors: bonventre jv. Am J Physiol Renal Physiol. 2006 Jan;290(1):F70-9. doi: 10.1152/ajprenal.00358.2004. Epub 2005 Jun 28. Am J Physiol Renal Physiol. 2006. PMID: 15985650 Free article.

Kidney Int 52: 294-308, 199). cPLA2-interacting protein (PLIP), the Tip60 splice variant, interacts with cPLA2 and enhances the susceptibility of renal mesangial cells to serum deprivation-induced apoptosis (Sheridan AM, Force T, Yoon HJ, O'Leary E, Choukroun G, Taheri MR, and …

Kidney Int 52: 294-308, 199). cPLA2-interacting protein (PLIP), the Tip60 splice variant, interacts with cPLA2 and enhances the susceptibili …

10

Restoration of tubular epithelial cells during repair of the postischemic kidney occurs independently of bone marrow-derived stem cells.

Duffield JS, Park KM, Hsiao LL, Kelley VR, Scadden DT, Ichimura T, Bonventre JV. Duffield JS, et al. Among authors: bonventre jv. J Clin Invest. 2005 Jul;115(7):1743-55. doi: 10.1172/JCI22593. Epub 2005 Jun 2. J Clin Invest. 2005. PMID: 16007251 Free PMC article.

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