Buttini M - Search Results

1

Increased T cell recruitment to the CNS after amyloid beta 1-42 immunization in Alzheimer's mice overproducing transforming growth factor-beta 1.

Buckwalter MS, Coleman BS, Buttini M, Barbour R, Schenk D, Games D, Seubert P, Wyss-Coray T. Buckwalter MS, et al. Among authors: buttini m. J Neurosci. 2006 Nov 1;26(44):11437-41. doi: 10.1523/JNEUROSCI.2436-06.2006. J Neurosci. 2006. PMID: 17079673 Free PMC article.

2

Beta-amyloid immunotherapy prevents synaptic degeneration in a mouse model of Alzheimer's disease.

Buttini M, Masliah E, Barbour R, Grajeda H, Motter R, Johnson-Wood K, Khan K, Seubert P, Freedman S, Schenk D, Games D. Buttini M, et al. J Neurosci. 2005 Oct 5;25(40):9096-101. doi: 10.1523/JNEUROSCI.1697-05.2005. J Neurosci. 2005. PMID: 16207868 Free PMC article.

3

Neuron-specific apolipoprotein e4 proteolysis is associated with increased tau phosphorylation in brains of transgenic mice.

Brecht WJ, Harris FM, Chang S, Tesseur I, Yu GQ, Xu Q, Dee Fish J, Wyss-Coray T, Buttini M, Mucke L, Mahley RW, Huang Y. Brecht WJ, et al. Among authors: buttini m. J Neurosci. 2004 Mar 10;24(10):2527-34. doi: 10.1523/JNEUROSCI.4315-03.2004. J Neurosci. 2004. PMID: 15014128 Free PMC article.

4

Mice as models: transgenic approaches and Alzheimer's disease.

Games D, Buttini M, Kobayashi D, Schenk D, Seubert P. Games D, et al. Among authors: buttini m. J Alzheimers Dis. 2006;9(3 Suppl):133-49. doi: 10.3233/jad-2006-9s316. J Alzheimers Dis. 2006. PMID: 16914852 Review.

5

Neutralization of soluble, synaptotoxic amyloid β species by antibodies is epitope specific.

Zago W, Buttini M, Comery TA, Nishioka C, Gardai SJ, Seubert P, Games D, Bard F, Schenk D, Kinney GG. Zago W, et al. Among authors: buttini m. J Neurosci. 2012 Feb 22;32(8):2696-702. doi: 10.1523/JNEUROSCI.1676-11.2012. J Neurosci. 2012. PMID: 22357853 Free PMC article.

6

Morphological characterization of Thioflavin-S-positive amyloid plaques in transgenic Alzheimer mice and effect of passive Abeta immunotherapy on their clearance.

Bussière T, Bard F, Barbour R, Grajeda H, Guido T, Khan K, Schenk D, Games D, Seubert P, Buttini M. Bussière T, et al. Among authors: buttini m. Am J Pathol. 2004 Sep;165(3):987-95. doi: 10.1016/s0002-9440(10)63360-3. Am J Pathol. 2004. PMID: 15331422 Free PMC article.

7

Expression of human apolipoprotein E3 or E4 in the brains of Apoe-/- mice: isoform-specific effects on neurodegeneration.

Buttini M, Orth M, Bellosta S, Akeefe H, Pitas RE, Wyss-Coray T, Mucke L, Mahley RW. Buttini M, et al. J Neurosci. 1999 Jun 15;19(12):4867-80. doi: 10.1523/JNEUROSCI.19-12-04867.1999. J Neurosci. 1999. PMID: 10366621 Free PMC article.

8

Elimination of the class A scavenger receptor does not affect amyloid plaque formation or neurodegeneration in transgenic mice expressing human amyloid protein precursors.

Huang F, Buttini M, Wyss-Coray T, McConlogue L, Kodama T, Pitas RE, Mucke L. Huang F, et al. Among authors: buttini m. Am J Pathol. 1999 Nov;155(5):1741-7. doi: 10.1016/S0002-9440(10)65489-2. Am J Pathol. 1999. PMID: 10550330 Free PMC article.

9

Modulation of Alzheimer-like synaptic and cholinergic deficits in transgenic mice by human apolipoprotein E depends on isoform, aging, and overexpression of amyloid beta peptides but not on plaque formation.

Buttini M, Yu GQ, Shockley K, Huang Y, Jones B, Masliah E, Mallory M, Yeo T, Longo FM, Mucke L. Buttini M, et al. J Neurosci. 2002 Dec 15;22(24):10539-48. doi: 10.1523/JNEUROSCI.22-24-10539.2002. J Neurosci. 2002. PMID: 12486146 Free PMC article.

10

Dominant negative effects of apolipoprotein E4 revealed in transgenic models of neurodegenerative disease.

Buttini M, Akeefe H, Lin C, Mahley RW, Pitas RE, Wyss-Coray T, Mucke L. Buttini M, et al. Neuroscience. 2000;97(2):207-10. doi: 10.1016/s0306-4522(00)00069-5. Neuroscience. 2000. PMID: 10799751

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