Selbach M - Search Results

1

Robust Salmonella metabolism limits possibilities for new antimicrobials.

Becker D, Selbach M, Rollenhagen C, Ballmaier M, Meyer TF, Mann M, Bumann D. Becker D, et al. Among authors: selbach m. Nature. 2006 Mar 16;440(7082):303-7. doi: 10.1038/nature04616. Nature. 2006. PMID: 16541065

2

Identification of a tyrosine-phosphorylated 35 kDa carboxy-terminal fragment (p35CagA) of the Helicobacter pylori CagA protein in phagocytic cells: processing or breakage?

Moese S, Selbach M, Zimny-Arndt U, Jungblut PR, Meyer TF, Backert S. Moese S, et al. Among authors: selbach m. Proteomics. 2001 Apr;1(4):618-29. doi: 10.1002/1615-9861(200104)1:4<618::AID-PROT618>3.0.CO;2-C. Proteomics. 2001. PMID: 11681214

3

Phosphorylation of tyrosine 972 of the Helicobacter pylori CagA protein is essential for induction of a scattering phenotype in gastric epithelial cells.

Backert S, Moese S, Selbach M, Brinkmann V, Meyer TF. Backert S, et al. Among authors: selbach m. Mol Microbiol. 2001 Nov;42(3):631-44. doi: 10.1046/j.1365-2958.2001.02649.x. Mol Microbiol. 2001. PMID: 11722731 Free article.

4

Src is the kinase of the Helicobacter pylori CagA protein in vitro and in vivo.

Selbach M, Moese S, Hauck CR, Meyer TF, Backert S. Selbach M, et al. J Biol Chem. 2002 Mar 1;277(9):6775-8. doi: 10.1074/jbc.C100754200. Epub 2002 Jan 11. J Biol Chem. 2002. PMID: 11788577 Free article.

5

Functional analysis of the Helicobacter pylori cag pathogenicity island reveals both VirD4-CagA-dependent and VirD4-CagA-independent mechanisms.

Selbach M, Moese S, Meyer TF, Backert S. Selbach M, et al. Infect Immun. 2002 Feb;70(2):665-71. doi: 10.1128/IAI.70.2.665-671.2002. Infect Immun. 2002. PMID: 11796597 Free PMC article.

6

cag+ Helicobacter pylori induces homotypic aggregation of macrophage-like cells by up-regulation and recruitment of intracellular adhesion molecule 1 to the cell surface.

Moese S, Selbach M, Meyer TF, Backert S. Moese S, et al. Among authors: selbach m. Infect Immun. 2002 Aug;70(8):4687-91. doi: 10.1128/IAI.70.8.4687-4691.2002. Infect Immun. 2002. PMID: 12117984 Free PMC article.

7

The Helicobacter pylori CagA protein induces cortactin dephosphorylation and actin rearrangement by c-Src inactivation.

Selbach M, Moese S, Hurwitz R, Hauck CR, Meyer TF, Backert S. Selbach M, et al. EMBO J. 2003 Feb 3;22(3):515-28. doi: 10.1093/emboj/cdg050. EMBO J. 2003. PMID: 12554652 Free PMC article.

8

Helicobacter pylori induces AGS cell motility and elongation via independent signaling pathways.

Moese S, Selbach M, Kwok T, Brinkmann V, König W, Meyer TF, Backert S. Moese S, et al. Among authors: selbach m. Infect Immun. 2004 Jun;72(6):3646-9. doi: 10.1128/IAI.72.6.3646-3649.2004. Infect Immun. 2004. PMID: 15155677 Free PMC article.

9

The Helicobacter pylori CagA protein induces tyrosine dephosphorylation of ezrin.

Selbach M, Moese S, Backert S, Jungblut PR, Meyer TF. Selbach M, et al. Proteomics. 2004 Oct;4(10):2961-8. doi: 10.1002/pmic.200400915. Proteomics. 2004. PMID: 15378755

10

Subproteomes of soluble and structure-bound Helicobacter pylori proteins analyzed by two-dimensional gel electrophoresis and mass spectrometry.

Backert S, Kwok T, Schmid M, Selbach M, Moese S, Peek RM Jr, König W, Meyer TF, Jungblut PR. Backert S, et al. Among authors: selbach m. Proteomics. 2005 Apr;5(5):1331-45. doi: 10.1002/pmic.200401019. Proteomics. 2005. PMID: 15717330

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