Kroemer G - Search Results

1

NF-kappaB constitutes a potential therapeutic target in high-risk myelodysplastic syndrome.

Braun T, Carvalho G, Coquelle A, Vozenin MC, Lepelley P, Hirsch F, Kiladjian JJ, Ribrag V, Fenaux P, Kroemer G. Braun T, et al. Among authors: kroemer g. Blood. 2006 Feb 1;107(3):1156-65. doi: 10.1182/blood-2005-05-1989. Epub 2005 Oct 13. Blood. 2006. PMID: 16223780 Free article.

2

Differential involvement of Th1 and Th2 cytokines in autoimmune diseases.

Kroemer G, Hirsch F, González-García A, Martínez C. Kroemer G, et al. Autoimmunity. 1996;24(1):25-33. doi: 10.3109/08916939608995354. Autoimmunity. 1996. PMID: 8937685 Free article. Review.

3

Mitochondrial permeability transition is a central coordinating event of apoptosis.

Marchetti P, Castedo M, Susin SA, Zamzami N, Hirsch T, Macho A, Haeffner A, Hirsch F, Geuskens M, Kroemer G. Marchetti P, et al. Among authors: kroemer g. J Exp Med. 1996 Sep 1;184(3):1155-60. doi: 10.1084/jem.184.3.1155. J Exp Med. 1996. PMID: 9064332 Free PMC article.

4

Lonidamine triggers apoptosis via a direct, Bcl-2-inhibited effect on the mitochondrial permeability transition pore.

Ravagnan L, Marzo I, Costantini P, Susin SA, Zamzami N, Petit PX, Hirsch F, Goulbern M, Poupon MF, Miccoli L, Xie Z, Reed JC, Kroemer G. Ravagnan L, et al. Among authors: kroemer g. Oncogene. 1999 Apr 22;18(16):2537-46. doi: 10.1038/sj.onc.1202625. Oncogene. 1999. PMID: 10353597

5

Mitochondrion as a novel target of anticancer chemotherapy.

Costantini P, Jacotot E, Decaudin D, Kroemer G. Costantini P, et al. Among authors: kroemer g. J Natl Cancer Inst. 2000 Jul 5;92(13):1042-53. doi: 10.1093/jnci/92.13.1042. J Natl Cancer Inst. 2000. PMID: 10880547 Review.

6

Caspase-independent commitment phase to apoptosis in activated blood T lymphocytes: reversibility at low apoptotic insult.

Dumont C, Dürrbach A, Bidère N, Rouleau M, Kroemer G, Bernard G, Hirsch F, Charpentier B, Susin SA, Senik A. Dumont C, et al. Among authors: kroemer g. Blood. 2000 Aug 1;96(3):1030-8. Blood. 2000. PMID: 10910919 Free article.

7

Pre-processed caspase-9 contained in mitochondria participates in apoptosis.

Costantini P, Bruey JM, Castedo M, Métivier D, Loeffler M, Susin SA, Ravagnan L, Zamzami N, Garrido C, Kroemer G. Costantini P, et al. Among authors: kroemer g. Cell Death Differ. 2002 Jan;9(1):82-8. doi: 10.1038/sj.cdd.4400932. Cell Death Differ. 2002. PMID: 11803376

8

Platelet formation is the consequence of caspase activation within megakaryocytes.

De Botton S, Sabri S, Daugas E, Zermati Y, Guidotti JE, Hermine O, Kroemer G, Vainchenker W, Debili N. De Botton S, et al. Among authors: kroemer g. Blood. 2002 Aug 15;100(4):1310-7. doi: 10.1182/blood-2002-03-0686. Blood. 2002. PMID: 12149212 Free article.

9

Megakaryocyte polyploidization is associated with a functional gene amplification.

Raslova H, Roy L, Vourc'h C, Le Couedic JP, Brison O, Metivier D, Feunteun J, Kroemer G, Debili N, Vainchenker W. Raslova H, et al. Among authors: kroemer g. Blood. 2003 Jan 15;101(2):541-4. doi: 10.1182/blood-2002-05-1553. Epub 2002 Sep 5. Blood. 2003. PMID: 12393414 Free article.

10

Mitotic catastrophe constitutes a special case of apoptosis whose suppression entails aneuploidy.

Castedo M, Perfettini JL, Roumier T, Valent A, Raslova H, Yakushijin K, Horne D, Feunteun J, Lenoir G, Medema R, Vainchenker W, Kroemer G. Castedo M, et al. Among authors: kroemer g. Oncogene. 2004 May 27;23(25):4362-70. doi: 10.1038/sj.onc.1207572. Oncogene. 2004. PMID: 15048075

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