Quilici C - Search Results

1

Lyn-deficient mice develop severe, persistent asthma: Lyn is a critical negative regulator of Th2 immunity.

Beavitt SJ, Harder KW, Kemp JM, Jones J, Quilici C, Casagranda F, Lam E, Turner D, Brennan S, Sly PD, Tarlinton DM, Anderson GP, Hibbs ML. Beavitt SJ, et al. Among authors: quilici c. J Immunol. 2005 Aug 1;175(3):1867-75. doi: 10.4049/jimmunol.175.3.1867. J Immunol. 2005. PMID: 16034130

2

Sustained activation of Lyn tyrosine kinase in vivo leads to autoimmunity.

Hibbs ML, Harder KW, Armes J, Kountouri N, Quilici C, Casagranda F, Dunn AR, Tarlinton DM. Hibbs ML, et al. Among authors: quilici c. J Exp Med. 2002 Dec 16;196(12):1593-604. doi: 10.1084/jem.20020515. J Exp Med. 2002. PMID: 12486102 Free PMC article.

3

Perturbation of the CD4 T cell compartment and expansion of regulatory T cells in autoimmune-prone Lyn-deficient mice.

Tsantikos E, Quilici C, Harder KW, Wang B, Zhu HJ, Anderson GP, Tarlinton DM, Hibbs ML. Tsantikos E, et al. Among authors: quilici c. J Immunol. 2009 Aug 15;183(4):2484-94. doi: 10.4049/jimmunol.0804346. Epub 2009 Jul 20. J Immunol. 2009. PMID: 19620313

4

CTLA4Ig alters the course of autoimmune disease development in Lyn-/- mice.

Oracki SA, Tsantikos E, Quilici C, Light A, Schmidt T, Lew AM, Martin JE, Smith KG, Hibbs ML, Tarlinton DM. Oracki SA, et al. Among authors: quilici c. J Immunol. 2010 Jan 15;184(2):757-63. doi: 10.4049/jimmunol.0804349. Epub 2009 Dec 4. J Immunol. 2010. PMID: 19966213

5

Autoimmune disease in Lyn-deficient mice is dependent on an inflammatory environment established by IL-6.

Tsantikos E, Oracki SA, Quilici C, Anderson GP, Tarlinton DM, Hibbs ML. Tsantikos E, et al. Among authors: quilici c. J Immunol. 2010 Feb 1;184(3):1348-60. doi: 10.4049/jimmunol.0901878. Epub 2009 Dec 30. J Immunol. 2010. PMID: 20042579

6

Perturbed myelo/erythropoiesis in Lyn-deficient mice is similar to that in mice lacking the inhibitory phosphatases SHP-1 and SHIP-1.

Harder KW, Quilici C, Naik E, Inglese M, Kountouri N, Turner A, Zlatic K, Tarlinton DM, Hibbs ML. Harder KW, et al. Among authors: quilici c. Blood. 2004 Dec 15;104(13):3901-10. doi: 10.1182/blood-2003-12-4396. Epub 2004 Aug 31. Blood. 2004. PMID: 15339845 Free article.

7

Mice lacking three myeloid colony-stimulating factors (G-CSF, GM-CSF, and M-CSF) still produce macrophages and granulocytes and mount an inflammatory response in a sterile model of peritonitis.

Hibbs ML, Quilici C, Kountouri N, Seymour JF, Armes JE, Burgess AW, Dunn AR. Hibbs ML, et al. Among authors: quilici c. J Immunol. 2007 May 15;178(10):6435-43. doi: 10.4049/jimmunol.178.10.6435. J Immunol. 2007. PMID: 17475873

8

Gain- and loss-of-function Lyn mutant mice define a critical inhibitory role for Lyn in the myeloid lineage.

Harder KW, Parsons LM, Armes J, Evans N, Kountouri N, Clark R, Quilici C, Grail D, Hodgson GS, Dunn AR, Hibbs ML. Harder KW, et al. Among authors: quilici c. Immunity. 2001 Oct;15(4):603-15. doi: 10.1016/s1074-7613(01)00208-4. Immunity. 2001. PMID: 11672542 Free article.

9

Gain-of-function Lyn induces anemia: appropriate Lyn activity is essential for normal erythropoiesis and Epo receptor signaling.

Slavova-Azmanova NS, Kucera N, Satiaputra J, Stone L, Magno A, Maxwell MJ, Quilici C, Erber W, Klinken SP, Hibbs ML, Ingley E. Slavova-Azmanova NS, et al. Among authors: quilici c. Blood. 2013 Jul 11;122(2):262-71. doi: 10.1182/blood-2012-10-463158. Epub 2013 May 21. Blood. 2013. PMID: 23692855 Free article.

10

"Emergency" granulopoiesis in G-CSF-deficient mice in response to Candida albicans infection.

Basu S, Hodgson G, Zhang HH, Katz M, Quilici C, Dunn AR. Basu S, et al. Among authors: quilici c. Blood. 2000 Jun 15;95(12):3725-33. Blood. 2000. PMID: 10845903 Free article.

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