Quilici C - Search Results

1

Imbalanced gp130-dependent signaling in macrophages alters macrophage colony-stimulating factor responsiveness via regulation of c-fms expression.

Jenkins BJ, Grail D, Inglese M, Quilici C, Bozinovski S, Wong P, Ernst M. Jenkins BJ, et al. Among authors: quilici c. Mol Cell Biol. 2004 Feb;24(4):1453-63. doi: 10.1128/MCB.24.4.1453-1463.2004. Mol Cell Biol. 2004. PMID: 14749363 Free PMC article.

2

Hematopoietic abnormalities in mice deficient in gp130-mediated STAT signaling.

Jenkins BJ, Quilici C, Roberts AW, Grail D, Dunn AR, Ernst M. Jenkins BJ, et al. Among authors: quilici c. Exp Hematol. 2002 Nov;30(11):1248-56. doi: 10.1016/s0301-472x(02)00929-3. Exp Hematol. 2002. PMID: 12423677 Free article.

3

Gugasyan R, Quilici C, I ST, Grail D, Verhagen AM, Roberts A, Kitamura T, Dunn AR, Lock P. Gugasyan R, et al. Among authors: quilici c. J Cell Biol. 2002 Jul 8;158(1):115-25. doi: 10.1083/jcb.200112066. Epub 2002 Jul 1. J Cell Biol. 2002. PMID: 12093790 Free PMC article.

4

Mice lacking both G-CSF and IL-6 are more susceptible to Candida albicans infection: critical role of neutrophils in defense against Candida albicans.

Basu S, Quilici C, Zhang HH, Grail D, Dunn AR. Basu S, et al. Among authors: quilici c. Growth Factors. 2008 Feb;26(1):23-34. doi: 10.1080/08977190801987513. Growth Factors. 2008. PMID: 18365876

5

Mice lacking both granulocyte colony-stimulating factor (CSF) and granulocyte-macrophage CSF have impaired reproductive capacity, perturbed neonatal granulopoiesis, lung disease, amyloidosis, and reduced long-term survival.

Seymour JF, Lieschke GJ, Grail D, Quilici C, Hodgson G, Dunn AR. Seymour JF, et al. Among authors: quilici c. Blood. 1997 Oct 15;90(8):3037-49. Blood. 1997. PMID: 9376584 Free article.

6

Gain- and loss-of-function Lyn mutant mice define a critical inhibitory role for Lyn in the myeloid lineage.

Harder KW, Parsons LM, Armes J, Evans N, Kountouri N, Clark R, Quilici C, Grail D, Hodgson GS, Dunn AR, Hibbs ML. Harder KW, et al. Among authors: quilici c. Immunity. 2001 Oct;15(4):603-15. doi: 10.1016/s1074-7613(01)00208-4. Immunity. 2001. PMID: 11672542 Free article.

7

Perturbed myelo/erythropoiesis in Lyn-deficient mice is similar to that in mice lacking the inhibitory phosphatases SHP-1 and SHIP-1.

Harder KW, Quilici C, Naik E, Inglese M, Kountouri N, Turner A, Zlatic K, Tarlinton DM, Hibbs ML. Harder KW, et al. Among authors: quilici c. Blood. 2004 Dec 15;104(13):3901-10. doi: 10.1182/blood-2003-12-4396. Epub 2004 Aug 31. Blood. 2004. PMID: 15339845 Free article.

8

Mice lacking three myeloid colony-stimulating factors (G-CSF, GM-CSF, and M-CSF) still produce macrophages and granulocytes and mount an inflammatory response in a sterile model of peritonitis.

Hibbs ML, Quilici C, Kountouri N, Seymour JF, Armes JE, Burgess AW, Dunn AR. Hibbs ML, et al. Among authors: quilici c. J Immunol. 2007 May 15;178(10):6435-43. doi: 10.4049/jimmunol.178.10.6435. J Immunol. 2007. PMID: 17475873

9

Candida albicans can stimulate stromal cells resulting in enhanced granulopoiesis.

Basu S, Zhang HH, Quilici C, Dunn AR. Basu S, et al. Among authors: quilici c. Stem Cells Dev. 2004 Feb;13(1):39-50. doi: 10.1089/154732804773099245. Stem Cells Dev. 2004. PMID: 15068692

10

"Emergency" granulopoiesis in G-CSF-deficient mice in response to Candida albicans infection.

Basu S, Hodgson G, Zhang HH, Katz M, Quilici C, Dunn AR. Basu S, et al. Among authors: quilici c. Blood. 2000 Jun 15;95(12):3725-33. Blood. 2000. PMID: 10845903 Free article.

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